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ILLUSTRATIONS.” 
Prater I. Japanese soldier in the Hiroshima Kakke Hospital. Dry, atrophic beri- 
beri with advanced muscular atrophies, contractions, and pes equino- 
varus. 
II. Japanese soldier in the Hiroshima Kakke Hospital. Dry, atrophic beri- 
beri. Paresis of the extensor muscles of the arms. 
III. Native Filipino, prisoner in Bilibid Prison, Hospital B. Chronic, wet 
beriberi. Cidema of the legs and of the dorsum of the feet. 
IV. Three native Filipinos, prisoners in Bilibid Prison and patients in the 
Main Prison Hospital. Chronic, wet beriberi. Cidema of the legs 
and of the dorsum of the feet. 
V. Native Filipino, Constabulary private. Acute, wet beriberi. Great 
cedema of the legs and feet. 
VI. Fig. 1. Acute, pernicious beriberi (Necropsy No. 1547). Longitudinal 
section of the popliteal nerve; Weigert-Pal stain. Showing degenera- 
tion of the myelin sheath. Low magnification. 
Fig. 2, Acute, pernicious beriberi (Necropsy No. 1547). Longitudinal 
section of popliteal nerve; Weigert-Pal stain. Showing rosary forma- 
tion and honeycombing of myelin sheath. Medium magnification. 
VII. Fig. 1. Acute, pernicious beriberi (Necropsy No. 1547). Longitudinal 
section of popliteal nerve; Weigert-Pal stain. Showing various types 
of degeneration of the myelin sheath. Medium magnification. : 
Fig. 2. Acute, pernicious beriberi (Necropsy No. 1547). ‘Transverse 
section of popliteal nerve; Weigert-Pal stain. Showing varying 
degrees of degeneration of the myelin sheath. Medium magnification. 
VIII. Fig. 1. Subacute beriberi (Necropsy No. 1636). Longitudinal section 
of popliteal nerve; Weigert-Pal stain. Showing profound degenera- 
tion of the myelin sheath. Medium magnification. 
Fig. 2. Subacute, wet beriberi (Necropsy No. 913). Longitudinal sec- 
tion of .popliteal nerve. Hamatoxylin-eosin stain. Showing pro- 
liferation of neurolemma nuclei. Medium magnification. 
IX. Acute, wet beriberi (Necropsy No. 1225). Section of myocardium. 
Hematoxylin-eosin stain. Showing fragmentation and segmentation. 
Medium magnification. 
X. Fig. 1. Subacute, wet beriberi (Necropsy No. 913). Section of the myo- 
cardium. Hematoxylin-eosin stain. Showing loss of striation vac- 
uolation, and granular degeneration. Medium magnification. 
Fig. 2. Acute, pernicious beriberi (Necropsy No. 1595). Section of the 
liver. Hematoxylin-eosin stain. Showing fatty degeneration and 
necrosis in the central area of the liver lobule. Medium magnifica- 
tion. 
“The photographs of beriberi patients are from the author’s original plates, 
taken in Hiroshima, Japan, and in Manila, P. I. The microphotographs are by 
Charles Martin, photographer of this Bureau, from sections prepared and stained 
by F. H. Willyoung, of the Biological Laboratory, Bureau of Science. 
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