12 Philippine Journal of Science 1920 
TABLE 6.—Cases not parasitized. 
[R, recovered; I, improved; D, died.] 
Case.) Age. | Sex. Diagnosis. pin ioe 
oe yr e eeu ese So aes Speers eee eer — 
Mos. | 
3 24} ¢& | Meocolitis with secondary bronchitis; severe_____..-.-_.------------...- R 
5 le} 9} Heoeohtia: moderately tevere —-<. 2025s Sa eee I 
32 22} @ | Bronchial asthma; rachitis; indigestion. (Vomited an ascarid before I 
admission.) 
76> 4 Seon tes SOCOnUeSy AOGIMMMUILIN. oo i eeeeue D 
| 57 Beh tor eatiors ee ec eee R 
SORE Sse Fs Bees ee re Se es aa ed R 
|. 8 18} co | Bronchopneumonia; moderately severe __........--...-..-..------------ I 
100 12} 2 | Malaria with secondary splenomegaly _..-..-..__.-.-_---..---------_--- I 
that has just been mentioned. Strangely enough, not one of 
these was infected with protozoa sensu stricto. The data on 
these cases are presented in Table 7. 
The age distribution and incidence of the several parasites 
together with the number of cases in each group are presented 
in Table 8. 
Inspection of Table 8 will show that infection with Spiro- 
cheta eurygyrata, “Blastocystis,” Trichuris, and Ascaris takes 
place exceedingly early in the life of the child, and these parasites 
are found in children of every age up to our limit of 13 years. 
Hookworm infections in Manila and the vicinity apparently do 
not get a fair start much before the sixth year, only an isolated 
ease having been picked up between the third and fourth years. 
Protozoal infections begin to take place after the first year and 
are more or less uniformly distributed through the series there- 
after. 
Tables 9 to 21 summarize the laboratory and clinical data 
throughout the entire series. It has been found interesting to 
break the series up into thirteen groups representing each year 
of the child’s life, for it was felt that such an arrangement 
might form a useful basis for comparison in later work. The 
parasites recorded represent only those found in our laboratory 
examinations, without reference to the passage or vomiting of 
worms before the child entered the hospital, or to the cases 
where children vomited or passed ascarids in the ward when 
laboratory examination had failed to discover the ova. There 
were only two such instances. 
The clinical diagnosis is that made in the ward and does 
not necessarily include the laboratory diagnosis. It is mainly 
a record of the salient features of the diagnosis as made when 
