2297921-5 



ANIMAL, CETACEA DOLPHIN 

 90092184 SBT 



Dr. Raymond J. Tarpley 

 Dept. of Veterinary Anatomy 

 Texas A&M University 

 College Station, TX 77843 



CPU-V 

 JMP/TPL/mab 



25 September 1990 



90092184 



1. Lung: Edema, alveolar, diffuse, moderate, with alveolar 



histiocytosis, Atlantic bottlenose dolphin ( Tursiops 



truncatus ), cetacean. 



T. Lung: Pyogranulomas, multifocal, moderate. 



3. Lung: Fibrosis, multifocal, moderate. 



4. Pancreas: Fibrosis and acinar cell atrophy, diffuse, 

 severe, with mild multifocal chronic and chronic -active 

 pancreatitis. 



5. Liver: Vacuolar change, microvesicular, diffuse, moderate. 



6. Liver: Sinusoidal dilatation, multifocal, moderate. 



7. Lymph nodes: Lymphoid atrophy, diffuse, mild. 



8. Thymus: Atrophy, diffuse, mild. 



Comment: Postmortem autolysis hindered microscopic evaluation. The pulmonary 

 edema may have been agonal or caused by drowning. The pulmonary pyogranulomas 

 were probably caused by metazoan parasites. The fibrosis in the lungs probably 

 represents areas of resolved pneumonia. The caM«;e of the severe pancreatic 

 fibrosis and atrophy was not apparent. Lesions of this type have been recognized 

 previously, but generally in older dolphins. It is likely that both exocrine and 

 endocrine pancreatic function were impaired. The cause of this lesion is 

 unknown. Hepatic vacuolar change can be caused by a number of different factors. 

 Microvesicular fatty change, which this vacuolar change resembles, has been 

 associated with toxic hepatic injury in some species. The thymic atrophy may 

 represent a normal physiologic process. The causes of the other lesions were not 

 apparent. 



JOHN M. PLETCHER, DVM, MPH 



Colonel, VC, USA 



Chairman, Department of Veterinary Pathology 



THOMAS P. LIPSCOMB, DVM 



MAJ, VC, USA 



Department of Veterinary Pathology 



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