Salt accumulation and mode of action of auxin 



pectin-free walls of fvmgi are apparently not affected by lAA. A more 

 extensive survey of I AA-stimulated cell walls is now projected; preliminary 

 results suggest that pectin-rich walls are the type susceptible to stimulation 

 by lAA. 



It is emphasized that this px'eliminary hypothesis is an attempt to stimulate 

 or open up new lines of research. As yet the secondary effects due to auxin 

 have hardly been considered, though it may be remarked that the high 

 concentrations required to induce bud dormancy may possibly be esterase 

 or accumulation-inhibiting concentrations. Interference with abscission 

 may be also connected with pectin metabolism. 



ACKNOWLEDGEMENTS 



I wish to thank Miss J. Turnham and Dr. B. Hazzi, who provided some of 



the experimental data quoted. 



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 Greig, M. E., Faulkner, J. S., and Mayberry, T. C. (1953). Studies on perme- 

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291 



