No. 2, Makch, 1921] PATHOLOGY 187 



of Mycology to work along similar lines to the already existing Imperial Bureau of Ento- 

 mology, for the identification of fungi, the publication of pathological literature, the estab- 

 lishment of a plant-pathology library, and the answering of enquiries of plant pathologists. — 

 J . S. Dash. 



1262. BoKURA, Umenojo. A new bacterial disease of gypsy-moth caterpillars. Ann. 

 Phytopath. Soc. Japan V: 34-55. 1920.— [Following ab.stract is from English resume, p. 

 55.) As a natural enemy of the gypsy-moth caterpillar {Lymanthria Dispar L.) this disease 

 which is wide-spread in Japan is, together with a parasitic species of Hemiptera, playing a 

 most important role in checking the spread of the insect. Appearing when the worm has 

 reached its fourth or fifth moulting skin, the disease, "Nankwabyo," resembles the "Nank- 

 wabyo," a kind of "flacherie" of silk worms. It is caused by a new species of bacteria, 

 Bacillus Disparis Hori and Bokura. The pathogene is rod-shaped, measures 1-1.3 X 0.7- 

 0.8 microns, with 6 to 8 peritrichiate flagella. Endospores are not formed, and it is not dis- 

 colored by the Gram's method. Konjak-medium is not liquefied. Fermentation is readily 

 set up in dextrose, laevulose, saccharose, maltose, and galactose. Optimum temperature is 

 30°-32*'C. When fed to gypsy-moth caterpillars death ensues in from 1 to 8 days. It is 

 non-pathogenic to silkworms, Anomala rrifocuprea Motsch., Pieris rapae L., Aphis sp. on 

 lily and Hemerophila atrilineata Butl. on mulberry. It is recommended that sterilized water 

 in which the bouillon or soy-bean cake decoction culture of the bacteria is mixed be sprayed 

 on plants on which the worms are feeding, or else the bodj'-fluid of the diseased caterpillars 

 be used as spray material. — L. M. Massey. 



1263. Dickson, B. T. Report of the delegates to the Canadian Branch of the American 

 Phytopathological Society. Ann. Rept. Quebec Soc. Protection of Plants 12: 24-27. 1920. — 

 An account of the annual meeting held on December 11 and 12, 1919, at the Ontario Agric. 

 Coll. A short summary of each address and paper is given. — B. T. Dickson. 



1264. HoRT, Edward C. The cultivation of aerobic bacteria from single cells. Jour. 

 Hygiene 18:361-368. 1 fig. 1920. 



1265. HoxiE, Frederick J. Roof construction for factories with excessive moisture. 

 Amer. Architect 115: 181-187. 20 fig. Jan. 29, 1919.— Rotting of roof planks and sweating 

 can be prevented in a large part by improved design and construction. Chamfering off 

 upper corners of wide beams would prevent the usual rotting off of roof planks above the 

 beams. Ornamental moldings at the upper edges of the beams, and sheathing should be 

 avoided. Insulation upon the outside of a roof should prevent precipitation of water within 

 the plank. Such an insulated roof would consist of an outer creosoted plank and an inner 

 untreated plank separated by waterproofed material. Insulation added to old roofs which 

 have begun to decay should retard the destruction. Other means of assistance in preventing 

 the rot in roofs are: insulation of skylights, drains, and ventilators to prevent radiation of 

 heat; the proper placing of heating pipes to keep the roof dry and warm, and prevent circu- 

 lation of moist air against cool roofs. — Walter H. Snell. 



1266. HuDiG, J., AND C. Meyer. De Hooghalensche ziekte een nieuwe bodemziekte on 

 Zand — en veengronden. [The Hooghalen disease, a new soil disease on sand and peat soils.] 

 25 p. Den Haag: J. en H. van Langenhuysen, 1918. 



1267. Jones, L. R., M. Miller, and E. Bailey. Frost necrosis of potato tubers. Wis- 

 consin Agric. Exp. Sta. Res. Bull. 46. 46 p., 1 colored pi., 12 fig. 1919.— Potatoes slightly 

 frosted may be internally discolored when seemingly unaffected externally. Such injuries 

 are termed "frost necrosis" and should be differentiated from other necrotic lesions, especially 

 the non-parasitic but inheritable "net necrosis" and the parasitic types of "ring necrosis." 

 The symptoms, which are described and figured in detail, include blotching and netted or ring 

 discolorations of the interior tissues, and are more pronounced at the stem end than eye end 

 of the tuber and in the vascular elements more than in the parenchyma. Experimental 



