238 PATHOLOGY [Bot. Absts, 



In infested soils, liberal application of fertilizers, especially those rich in nitrogen and 

 humus, aids in producing fairly normal yields. Author first reported the disease in Phyto- 

 path. 7:59. 1919.— A. H. Chivers. 



1664. Ttjnstall, A. C. A stem disease of tea caused by Nectria cinnabarina (Tode) Fr. 

 6 p., 4 pi- Indian Tea Assoc: Calcutta, 1918. — The diseased bushes become moribund, but 

 rarely die outright. The stems die back, and new shoots, generally thin and weak, spring 

 up from below. The bark, cambium, medullary rays, pith, and wood are affected, the fungus 

 apparently gaining entrance through wounds. Descriptions of the fungus by the author, by 

 Wilson and Seaver, and from Saccardo are given, also a list of synonyms by Seaver. — The 

 flowering shoots of Alnus nepalensis, Pyrularia edulis, and Erythrina are attacked by the 

 fungus and serve as sources of inoculum. Control measures recommended are: eradication 

 of trees (in immediate neighborhood) harboring the fungus; pruning diseased bushes in 

 "cold weather," followed by spraying with a fungicide; and burning of prunings. [See 

 Bot. Absts. 3, Entry 1199.] — J. I. Lauritzen. 



1665. Turconi, M. Un nuovo parassita dei peperoni (Acrothecium Capsici n. sp.) [A 

 new parasite of pepper.] Revist. Patol. Veg. 9: 131-133. 1919. — In examining some peppers 

 {Capsicum annuum) sent from the Royal normal school of Turin some were found with yel- 

 lowish white areas, which later became dark-colored, due to the fruiting bodies of Alter- 

 naria tenuis, a common saprophyte. There were other round or oval depressed spots, 1-3 

 centimeters in diameter, of hazel color with chestnut brown margins, in which appeared a 

 thin, olivaceous yellow mold which the author names Acrothecium capsici. A technical 

 description is given.— F. M. Blodgett. 



1666. van der Lek, H. A. A. Over de z. g. verwelkingsziekten, in het bijzondef die, 

 welke door Verticillium alboatrum veroorzaakt worden. [Regarding the so-called wilt diseases, 

 especially those caused by Verticillium alboatrum.] Tijdschr. Plantenz. 24: 205-219. PI. 4, 

 fig. 1-3. 1918. Ibid. 25: 17-52. PI. 1-2, fig. 1-4. 1919.— In an introduction of 14 pages, the 

 author first records an exceptionally severe outbreak of Verticillium wilt of potatoes in 

 Holland in 1918, and also its occurrence in tomatoes, cucumbers and melons. He also pre- 

 sents a rather detailed and critical review of the literature dealing with Verticillium dis- 

 eases, examining especially the evidence on host range, biological strains, identity of Ver- 

 ticillium and Acrostalagmus species, the disease in woody plants, and the peculiar position of 

 the potato as a host of Verticillium. In this last connection he calls attention to the re- 

 corded inability of Verticillium strains from perennial hosts to infect annuals, while strains 

 from the latter readily attack the former. This he attributes to a loss of virulence due to 

 continuous confinement to a perennial host. He here reports for the first time isolation of 

 Verticillium from Thuya, Prunus (cherry), Ampelopsis veitchii and Ribes (currant). — 

 Parts II, III and IV in volume 25. They are devoted respectively to a consideration of: 

 variations in the symptoms of the disease in the same and different hosts; the potato verti- 

 cilliose in particular; and dissemination and methods of combating the pathogene. An 

 addenda presenting some observations on papers appearing after this article went to press 

 completes the contribution. — After discussing the physiology of wilting in plants the author 

 points out that this is rather rare as a symptom of Verticillium diseases. When it does 

 occur he holds it is due to a killing of the rootlets growing in infested soil. Plants so affected 

 usually recover shortly to show later the more common symptoms; dwarfing, dead spots with 

 yellowish borders in the leaves or the slow dying of leaves on the stems from below upwards. 

 In his opinion wilting rarely or never occurs in potato plants infected from diseased mother 

 tubers. He rejects the theory of wilting due to toxic substances secreted by the fungus, as 

 also that of the plugging of the vessels by the mycelium. — In part three is presented evidence 

 to support the conclusions that diseased mother tubers give usually infested daughter tubers, 

 while healthy tubers from diseased plants never produce diseased plants; that the fungus 

 spreads slowly during storage from the stem end through the vessels toward the eyes; that 

 the variations in symptoms so often noted for this disease especially in potatoes, is to be ex- 



