TUNEVALL AND LINDNER 



did this. I was in Colorado last March and talked with one of 

 his young gentlemen. They have since repeated this work using 

 more moderate levels of cooling — 30° C — and the opposite 

 result was obtained. There was a much more significant rate 

 of survival at the more moderate level of cooling, and this ob- 

 viously related to the host problem that I mentioned. 



TUNEVALL: My characterization of the situation relates to 

 the early 1950's when hypothermia was already being used ther- 

 apeutically in several clinical conditions. I don't think they knew 

 very much what they were doing then, 



BLAIR: I discussed it a few moments ago. They used arti- 

 ficial hibernation, and I hate those words, 



CAMPBELL: Do you think new immunomechanisms, antibodies, 

 play any part in it? 



TUNEVALL: In this connection, no, I don't think so, 



CAMPBELL: But there aren't any so-called natural antibodies 

 on the antigens that you were testing? 



BERRY: In that connection, at the meetings in Montreal it 

 was said by one of the speakers on a symposium that all ani- 

 mals have a very high immunity, not a natural immunity. It 

 is acquired through contact with staphylococci and this is the 

 reason why any immunization against staphylococci is so un- 

 successful. They are already maximumally immunized. This 

 is a concept that never occurred to me, but maybe it has some 

 validity, at least in regard to the staphylococcin antibodies 

 not normally present as a result of the contact. 



TUNEVALL: I think that the only way to bring active anti- 

 body formation into this picture wouldbe to arrange for a secondary 

 response. 



4 Dr. David Rogers, VanderbuiU University. 



150 



