160 PHYSIOLOGY OF INDUCED HYPOTHERMIA 



TABLE II 



Cardiovascular and Respiratory Dynamics in Dogs Cooled After Induction 

 OF Hemorrhagic Shock 



« g'CJ 



Prior to cooling 



or shock 38-39 



During shock 38-39 



During hypothermia 



and shock 24 30 84 366 17.6 14.2 15.5 68.8 21 



After transfusion 



and rise in body 



temperature 33.2 



Number of experiments 



for each value listed — 



mal values in all categories. The notable increase above normal in oxygen consump- 

 tion is perhaps due to greater muscular activity than during the initial period of 

 observation under morphine. 



Comparison of the foregoing data with those in dogs cooled after the induction 

 of shock (table II) reveals several points of interest. The fall in oxygen consump- 

 tion and cardiac output is appreciably less than in the precooled dog and the respira- 

 tory depression is not nearly so steep. The higher level of metabolic activity, as 

 reflected in the higher oxygen consumption, suggests that when the circulation is 

 already defective external cooling may fail to reduce the temperature of some of the 

 deeper tissues to the same level as in the precooled animal. 



