151. O^ CONSUMPTION: ANIMAL TISSUES (Continued) 



Part VUI: NERVE TISSUES (Concluded) 



Section 1: Central and Retinal (Concluded) 



K., ibidrn;381, 421, 1926. (31] Warburg, O., Posener, K., and Negelein, E., ibid 1^:309, 1924. [32] Peiss, 

 C. N., and Field, J.. Arch. Biochem. and Biophys. 36:276, 1952. [33] Hoexter, F. M., Endocrinology 54: 1 , 1954. 

 [34] Pearce, J., and Gerard. R. W., Am. J. Physiol. n6:49, 1942. [35] Roberts, $., and Rock, M., unpublished. 

 [36] Roberts, S., and Keller, M. R., Arch. Biochem. and Biophys. 44:9. 1953. [37] SeUei, C, Weinstein, P., and 

 Jany, J., Biochem. Zschr. 247:146, 1932. [38] Negelein, E., ibid 165:122, 1925. [39] Greig, M. E., Munro, M. P., 

 and Elliot, K. A., Biochem. J., Lond. 33:443. 1939. [40] Laser, H., ibid 31^: 1 67 1 . 1937. [41] Roe. C, Acta Ophth. , 

 32:181, 1954. [42] llling, E. K., and Gray, C. H., Endocrinology 7:242, 1951. [43] Robbie, W. A., and Leinfelder, 

 P. J.. Am. J. Ophth. 32:208, 1949. [44] EUiott, K. A., and Baker, Z., Biochem. J., Lond. 29:2433, 1935. 



Section 2: Peripheral 



Values for oxidation quotient (QO2) are expressed in cu ram oxygen consumed per g fresh weight of tissue per hour. 

 Values in parentheses are ranges, estimate "c" of the 95% range (cf Introduction). 



III Using oxygen electrode. /2/ In blood; reduced to 41 at 170C. /3/ Corrected for temperature; mean R. Q., 0.83 

 (0.77-0.88). /4/ In winter; increased to 21 in spring; steady for 20 hr. /5/ Mostly in winter frogs; increased by 

 half in summer; Qjg was 2.2. /6/ Figures calculated from Fenn's data by Gerard (1932); increased to 56 in 

 summer. /7/ In winter; increased to 28 in spring; Qjo. 2.2. /8/ In winter. /9/ Reduced to 30 at 22°C; not altered 

 if degenerated for a week. /lO/ 235 in 2nd hr. /ll/ In blood; not significantly different in Tyrode; slightly higher 

 in calves; reduced to 68 at 17°C. /12/ In sea water. /13/ In sea water, decreases by 1.5% per hr. 



Contributors : (a) Vernberg. F. J., (b) Fitzgerald, L. R., (c) Keynes, R. D. 



References : [ l] Cardot, H., Faure, S., and Arvanitaki, A., J. physiol. path. g^n. 42:849, 1950. [2] Connelly, 

 C. M., Biol. Bull. 103:315, 1952. [3] Larrabee. M. G.. and Bronk, D. W., Cold Spring Harbor Symposia Quant. 

 Biol. r7:245, 1952.~T4] Rosenbaum, H., Biochem. Zschr. 247:189, 1932. (5) Fenn, W. O., Am. J. Physiol. 

 80:327, 1927. [6] Chang. T. H., Gerard, R. W., and Shaffer, M., ibid 101:19, 1932. [7] Gerard, R. W., ibid 

 82:381, 1927. [8] Gerard, R. W., Proc. Soc. Exp. Biol. 27:1052, 1930. T?] Fenn, W. O. , Am. J. Physiol. 92:349, 

 1930. [10] Meyerhof, O., and Schmitt, F. O., Biochem. Zschr. 208:445, 1929. [ll] Sherif. M. A., J. Pharm. 

 Exp. Ther. 38:231, 1930. [12] Gerard, R. W., and Mclntyre, M. D. (quoted by Gerard, R. W.), Physiol. Rev. 

 _12:469, 1932. [l3] Chang. T. H., Proc. Soc. Exp. Biol. 28:954, 1931. 



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