VETERINARY MEDICINE. 285 



which appear to be authentic. The biology of the parasite is discussed, together 

 with ail account of the course of tlae disease as it occurs in man. 



On some examinations of blood of the horse, ox, and sheep in Ma,dagascar, 

 J. Mamet and Loiselet [Rev. G6n. Med. Vit., 17 {1911), No. 198, Dp.. 330- 

 335). — The authors state that in Madagascar piroplasmosis occurs in the horse, 

 ox, and sheep. The bovine form, which is known to the natives as " beravy " or 

 " tazon'ouiby," is not considered to be of economic importance, as it always occurs 

 in a benign form. Nevertheless the authors think that its diminution of the 

 resistance of cattle to other diseases may explain the great annual loss caused in 

 Madagascar and Bara by tuberculosis and charbou, particularly the acute 

 course run (6 to 12 hours) by charbon in cattle. Equine piroplasmosis is more 

 grave than bovine and may prove fatal. This disease in sheep explains the 

 difficulty encountered in raising them in Madagascar. 



The species of tick or ticks that serve as active agents in the transmission of 

 these affections remain to be determined. 



On the development of Piroplasma parvum in the organs of cattle suf- 

 fering from East Coast fever, K. (Jonuer (Berlin. 'Ticrarztl. Wclinsclir., 26 

 (1910), No. 27, 537-589, /i(js. i7).— This account of TlicUeria (PiropUisnui) 

 parva has been previously noted from another source (E. S. R., 24, p. 83). 



The development of Theileria parva, the cause of East Coast fever of 

 cattle in Africa, R. Gonder (Arch. Protistenk., 21 (1910), No. 2, pp. 11,3-164^ 

 pis. 5, fig. 1). — This is a detailed report of the investigations noted above, with 

 references to the literature, of which a bibliography of 20 titles is appended. 



The prevention and eradication of East Coast fever, A. Theiler and J. M. 

 Christi' (Transvaal Dcpt. Agr., Farmers' Bui. 129, 1910, pp. 15, figs. 7 ; Trans- 

 vaal Ayr. Jour., 9 (1910), No. 38, pp. 1-3, figs. 7). — A general consideration of 

 the disease, the ticks which transmit it, and preventive and remedial measures. 



Trypanosome diseases of domestic animals in Uganda, IV, V, D. Bruce 

 ET AL. (Proc. Roy. Soc. [London], Ser. B. 83 (1911), No. B 563, pp 176-^186, 

 pis. 3, figs. 2). — Continuing i)revious work (E. S. R., 24, p. 586), the authors 

 find that Trypanosoma uniformc resembles T. vivax in shape and general 

 appearance but differs markedly in size and that it also resembles T. vivax in 

 not being pathogenic in the smaller laboratory animals. There is no evidence 

 available as to what is the carrier of T. uniforme. 



" T. nan/um is indistinguishable from T. pecorum either in the living condi- 

 tion or when fixed and stained. It differs from T. pecorum in not being patho- 

 genic to the smaller laboratory animals. The carrier of T. nanum is probably 

 the same as that of T. pecorum; as both diseases occur under the same condi- 

 tions, but there is no evidence available as to what the carrier is." 



Experiments to ascertain if antelope may act as a reservoir of the virus 

 of sleeping sickness (Trypanosoma gambiense), D. Bruce et al. (Proc. Roy. 

 Soc. [London], Ser. B, S3 (1911), No. B 564, pp. 311-327).— While antelope have 

 not been found naturally infected with T. gambiense, the authors have dis- 

 covered that water buck, bush buck, and reed buck can readily be infected with 

 a human strain of this trypanosome through the bites of infected Glossina 

 palpalis. 



"Antelope of the water buck, bush buck, and reed buck species, when infected 

 with the virus of sleeping sickness, can transmit the infection to clean labora- 

 tory-bred G. palpalis. This transmission of the infection to clean laboratory- 

 bred flies may occur at least 81 days after the last feed of the infected flies on 

 a buck. G. palpalis, when infected with the virus of sleeping sickness obtained 

 from the blood of infected antelope, are capable of transmitting the virus to 

 suggeptible j^uiBials." 



