VETERINARY MEDICINE. 1*79 



considerable danger in building up lu^rds with nonre:uiiii^' :iiiinials talcen from 

 herds in which there is tubercnlosis. 



The problem of bovine tuberculosis control, M. H. Hioynolds (Amer. Vet. 

 Rev., 3Ji [190i)\. X<>. .'/, pp. .1,1,9— 'iSl ) .—Thin is an address presented at the Inter- 

 national Congress on Tuberculosis, held at Washington, D. C. 



A review is given of the work against the disease in Minnesota, Pennsylvania, 

 Wisconsin, and Massachusetts. In the conclusions drawn from the study of 

 conditions in these States, the author considers that the more important ad- 

 vances made have been the education of the public and the enlistment of a 

 favorable opinion. In the discussion of eradication the author uses Minnesota 

 as an ilhistratiou of the i^eneral ])r()blem. A plan is suggested f(ir control work. 



The admission of Canadian store cattle into this country: What are the 

 dangers? H. A. Woodruff {Vet. Bee, 21 (1908), No. 1068, pp. J,n-419; 21 

 (1909). No. 1069, pp. 432, J,33). — In view of the demand for a revision of the 

 stringent regulations in respect to the admission of live cattle into Great 

 Britain from abroad, the author here considers the possible danger from free 

 importations from Canada. The diseases discussed are contagious bovine 

 pleuro-pHPunionia, foot-and-mouth disease, mange, tuberculosis, and Texas fever. 



Investig'ations on the occurrence and frequency of streptococcic mastitis 

 in cows, G. RuHM {Wch.nschr. TierJieilk. u. Viehzucht, 52 (1908), No. 7, pp. 

 125-130; (lbs. in Berlin. Tierartztl. Wchnscln:, 1908, No. 50, p. 902; Jour. Compar. 

 Path, and Ther., 22 {1909), No. 1, pp. 62, 63).— Aside from tubercle bacilli, 

 streptococci ai'e probably the pathogenic organisms most frequently found in 

 milk* They are often present, and since they are regarded by many medical 

 men as a cause of infant mortality, the consumption of milk containing them 

 must be considered dangerous for young children. 



Experiments were carried out by the author in order to test Trommsdorff's 

 method of determining the quantity of leucocytes present in milk. Five out of 

 16 cows in one stable were found to be affected with streptococcic mastitis. 

 The author considers the processes in the development of the disease to be as 

 follows: During the incubation period the streptococci increase in numbers 

 without, however, causing any injury to the substance of the udder. As the 

 bacterial products accumulate, however, inflammatory symptoms develop and 

 leucocytes are poured out in increased quantity. Whenever the leucocytes 

 increase in numbers to any great extent they immediately constitute an infal- 

 lible indication of commencing inflammation. As milking removes the strep- 

 tococci and their toxins, as w^ell as the leucocytes and the antitoxins, it prolongs 

 the course of the disease and renders the clinical symptoms less marked until 

 the afl'ected quarter gradually loses its function. 



Lung sickness or contagious pleuro-pneumonia of cattle, J. M. Christy 

 {Transvaal Apr. Jour., 7 {1909), No. 26, pp. 199-201) .—Nlthowgh. nearly every 

 South African farmer has had experience with this disease, the Transvaal is 

 to-day practically free from it. Cattle from a colony in which lung sickness 

 exists are not allowed to enter the Transvaal unless certified by a government 

 veterinary surgeon to be free from the disease and to come from a property 

 where contagion of animals has not existed for at least three months prior to 

 the date of such certification. A brief account is given of the occurrence of the 

 disease in other countries. 



Hemorrhagic spotting [cavernous angioma] of the liver in bovine animals, 

 P. Chausse {Rcc. Med. VH., 85 {1908), No. 21, pp. 101,-115, fiys. 2; ahs. in Jour. 

 Compar. Path, and Ther., 22 {1909), No. 1, pp. 56-6i).—" Hemorrhagic spot- 

 ting of the liver is common in cows, much rarer in oxen, and never occurs in 

 calves or in other animals slaughtered for meat. No theory hitherto advanced 

 regarding its pathogeny appears satisfactory. The ijrcdoniinance of the portal 



