790 EXPERIMENT STATION RECORD. 



infested by gourd worms shows that in these affections there is a chronic poison- 

 ing due to substances excreted by the parasites in the liver. 



Sheep dips, F. P. Sargeant (PJiunn. Jour. [London], If. sei:, 28 (1909), No. 

 2378, pp. 6Jt2, 643; abs. in Chem. ZcntU., 1909, II, No. 3, p. 231).— The author 

 discusses the various chemicals used to combat the parasites of the sheep fleece. 

 The different formulas and most efficient chemicals are enumerated. 



Recent work of the Bureau of Animal Industry concerning the cause and 

 prevention of hog cholera, :\I. Dorset ( [/. .S. Dept. Ayr. Yearbook 1908, pp. 

 321-332). — An account is given of hog cholera and of the methods of immunizing 

 hogs against the disease as worked out by this Department. 



Hog-cholera serum [Michigan 8ta. Circ. 6, pp. //, figs. 2). — Directions are 

 given for the use of hog-cholera serum which is now being manufactured at the 

 bacteriological laboratory. The serum-simultaneous method is not recommended 

 at present, but the serum-alone method is strongly recommended for use where 

 hog cholera exists or in eases where immunity is desired for a short period. A 

 charge of 2 cts. per cubic centimeter is made for the serum, which is said to be 

 below the actual cost for producing and testing it. A contract releasing the 

 laboratory from all i-esponsibility is required of all who use the serum. 



Swine fever (Agr. Gaz. N. 8. Wales, 20 (1900), No. S, pp. 656-669, flgs. 8).— 

 Hog cholera is said to have been known in New South Wales for about 6 years. 



Contribution to the knowledge of infectious anemia of horses, J. Hempel 

 (Ztschr. Infektionskrank. u. Hyg. Hausticre, 5 (1909), No. 5, pp. 3Sl--i34). — 

 The author finds the causative agent of this disease to be an ultravisible non- 

 culturable virus. It produces the disease when small quantities are subcu- 

 taneously or intravenously injected, but large quantities are necessary to pro- 

 duce it through ingestion. The saliva of an affected animal is not infectious 

 and healthy animals do not contract the disease through the inhalation of the 

 same air nor by eating from the same troughs as the diseased ones. An inter- 

 mittent fever is the only striking symptom. Recovered animals are not immune 

 to the disease and it is not possible to immunize animals against it. A con- 

 tinued heating for 1 hour at a tempei*ature of 56° C. does not destroy the virus. 



Equine infectious anemia, A. T. Kinsley (Amer. Vet. Rev., 36 (1909), No. 

 1, pp. J/S-SJf). — This paper is based on correspondence with 60 veterinarians 

 practicing in various localities where the disease occurs and on field investiga- 

 tions of about 50 cases in northern Oklahoma and southeastern Kansas. 



"The infection, in many instances, seems to remain on a farm or in a barn 

 indefinitely. It is usually not readily carried to adjacent farms or other barns. 

 A farm near Coft'eyville, Kans., has been infected for 12 or 14 years, and the 

 owner has lost horses with this disease nearly every year during that time, 

 while a neighbor living not more than 60 rods diagonally across a thoroughfare 

 has never had a case." 



Horses and mules were found to be about equally susceptible to the disease, 

 while age and breed appear to have little influence over susceptibility. Food 

 seems to have no causal relation to the disease, as it affects as many horses 

 that are constantly fed on dry food as those which run in pastures. The disease 

 occurs regardless of the source of water. 



"The initial attack of this disease usually occurs during July, August, and 

 September, though many cases have been observed both earlier and later than 

 this. The disease is most prevalent in the summer months; however, chronic 

 cases may be seen in midwinter. The disease is more prevalent during wet 

 seasons and is equally prevalent on uplands and lowlands. The disease may ap- 

 pear suddenly or it may have an insidious onset." The symptoms of the disease 

 are discussed at some length, and post-mortem examinations reported upon. 



" The most characteristic microscopic lesions were found in the blood. The 

 hemoglobin content, which according to the Tallquist scale ranges from 00 to 100 



