YETERINAEY SCIENCE AND PRACTICE. 505 



decolorize by Gram's method. Spore formation takes place after 18 hours' growth, 

 at a temperature of 37° C. The organism may be cultivated on all ordinary media 

 and growth takes place best at a temperature of 37° C. The bacilli are destroyed by 

 exposure to a temperature of 72° C. for 10 minutes, and the spores were killed by 

 being maintained at 100° C. for 5 minutes. Mice when inoculated with this bacillus 

 invariably died in less than 48 hours. The spleen was found enlarged, (iuinea pigs 

 inoculated with 1 cc. of a culture of the organism developed an inflammatory nodule 

 at the point of inoculation, or in some cases appeared to be infected. The organism 

 in question resembles closely the anthrax bacillus, but differs from it, however, in 

 its motility and its very limited pathogenic powers. 



Anthrax in dogs, Lupke {Dent. Tierdrztl. Wchnschr., 9 {1901), No. 4S, pp. 4S5- 

 489).— In the author's opinion the usual text-book statements concerning the prev- 

 alence and seriousness of anthrax in dogs is inadequate. The matter is frequently 

 treated as if anthrax was an exceedingly rare disease in dogs. Attention is called to 

 the frequency of the occurrence of anthrax in dogs and a number of examples are 

 cited where dogs became fatally infected from eating the meat of animals dead of 

 anthrax. The usual symptoms of anthrax in dogs are described in detail. 



Babies, D. E. Baughman {Amer. Yd. Rev. ,26 {1902), No. 2, pp. 115-123).— Ai\Br\t\oii 

 is called to the recent controversy in which the existence and nature of rabies have 

 been involved. The author gives a critical review of some of the more important 

 literature on the su1)ject, and accepts the view that rabies is a real disease, produced 

 by a virus which may be communicated by means of the bites of dogs and other 

 animals. 



Antitoxin in distemper, H. A. Stevenson {Jour. Comp. Med. and Vet. Arch., 23 

 {1902), No. 2, p. 116).— In a case of dog distemper, in which all of the usual symp- 

 toms were present and in which the ordinary remedies failed to produce the desired 

 result, Mulford's distemper antitoxin was used in doses of moderate size and caused 

 a decided improvement in the condition within 24 hours. After 48 hours the dis- 

 charge from the nose was checked and a complete recovery took place inside of one 

 week. 



Malignant jaundice in the dog, W. Robertson {.Toar. Comp. Path, and Ther., 14 

 {1901), No. 4, pp. 327-336, Jiga. 5). — The author describes in detail the symptoms of 

 this disease and briefly reviews the literature of the subject. Notes are given on the 

 post-morten lesions found in dogs affected with the disease and on the nature of the 

 blood parasite. Numerous remedies were tried without much success. The best 

 results were obtained from the administration of calomel and quinin. Evidence is 

 presented to show that the blood parasite which causes this disease is conveyed from 

 one animal to another through the agency of the dog tick {Hiemaphijsalis leachi). 



Intravenous injections of corrosive sublimate, A. Serafini {Pub. 1st. Vniv. 

 Padova,2 {1902), NIV, pp. 17; e.vtr. from Riforma Med., 18 {1902), No. 79-80).— 

 Exjieriments in intravenous injections of corrosive sublimate indicated that this 

 substance does not exercise a germicide action upon the organisms of anthrax and 

 fowl cholera in solution in the ratio to the weight of the body of 1: 360,000 or in the 

 ratio to the weight of the blood of 1 : 30,000. The author does not believe that this 

 method can become of general therapeutic value in the treatment of infectious 

 diseases. 



The causes which determine the absence of phagocytosis in the last 

 stages of fowl cholera in rabbits, B. Werigo {Arch. Med. Exper. et. Anat. Path., 

 Paris, 1. ser., 14 {1902), No. 2, pp. 156-202). — The author conducted a large number 

 of experiments, during which rabbits were inoculated with organisms of fowl 

 cholera. It was observed that during the last stages of the disease the phenomenon 

 of phagocytosis w'as absent. The author investigated the cause of this phenomenon 

 and concludes that the absence of phagocytosis in the last stages of the disease does 

 not depend upon any changes in the phagocyte cell. It is also believed that a 

 change of the medium or the saturation of the organic liquids with the bacterial 



