690 EXPERIMENT STATION RECOED. 



Immunization against African horse sickness, E. Leipziger (Ztschr. Infek- 

 tionskrank. «. Hyg. HausUere, 6 (1909), Nos. 1, pp. 52-80; 2, pp. 1-^3-179; ahs. 

 in Rev. Gen. Med. V6i., 15 (1910), ^o. 116, i)p. W, -i48).— An abstract of the 

 above. 



A new cat plague, A. Gaertner (Centbl. Bakt. [etc.], 1. Abt., Orig., 51 (1909), 

 No. 3, pp. 232-2Ji9). — In all of the cats which died during the epizootic the 

 author was able to isolate and cultivate the same liind of ovid rod bacterium 

 which belongs to the hemorrhagic septicemia group. When this rod bacterium 

 was introduced into the organism of the cat by inhalation, by inoculation, intra- 

 tracheally, or iutrathoracicly pneumopleuritis was produced, and from this 

 finding the author concludes that it is the cause of the cat plague. 



A new cat plague, Z. Skrzynski (CenthJ. Bakt. [etc.], 1. AM., Orig., 54 

 (1910), No. 5, p. ^51). — A polemical article in regard to the priority of thp above. 



[A new cat plague], A. Gaertner (Centbl. Bakt. [etc.], 1. Abt., Orig., 54 

 (1910), No. 5, pp. 451, 452). — A reply to the above, in which the differences in 

 the bacteria isolated are pointed out. 



Occurrence of acute anterior poliomyelitis in chickens, R. Wilke (Deiit. 

 Tieriirztl. Wcftnschr.. 17 (1909), No. -JT, pp. 697, 698; abs. in Centbl. Bakt. [etc.], 

 1. Abt., Ref., 46 (1910), No. 21, pp. 652, 653).— In an 8-day-old chick motor 

 paralysis occurred in both legs and wings but with sensory retention. In a 

 second case (0 to 8 weeks old) the motor paralysis also occurred but improved 

 gradually. Two other chickens S weeks old, from another source, showed the 

 same symptoms. The author attributes the malady to successive inbreeding, 

 which was the case with the above birds. The point of interest about these 

 cases is the occurrence of a number of cases of acute anterior poliomyelitis 

 amongst children in the same neighborhood. 



Role of the leucocytes in normal and immunized animals artificially in- 

 fected by the chicken cholera bacterium. A. Sulima (Ann. Inst. Pasteur, 23 

 (1909), No. 11, pp. 911-920). — Weyl's method (with bacteria-free exudates) is 

 deemed the best one to immunize guinea pigs and rabbits against fowl cholera. 

 This indicates that micro-organisms and their products thus received in the 

 animal body (which are the natural agressins) are better qualified as antigens 

 than the bacteria cultivated on an artificial media. If stained sections are 

 made of the subcutaneously inoculated areas of normal and immunized animals 

 it can be seen that a barrier of leucocytes is thrown about the locality of the 

 bacterial invasion. In the normal animal phagocytosis remains dormant, but 

 in immunized animals a moderate degree of this is evident in from 6 to 8 hours 

 and persists until the bacteria have all disappeared. On the other hand, normal 

 animals show a multiplication of the bacteria in their organism. 



The author assumes from this that owing to the sluggish manner in which the 

 phagocytosis goes on other humoral protective agents must be active at the same 

 lime. 



Contributions to our knowledge of leukemia, with particular reference to 

 this malady in chickens, Skiba (Dent. TivrarztJ. Wclmsehr., 17 (1909), No. 28. 

 pp. 405-412. figs. S). — A historical, clinical, and histopathological discussion of 

 the subject, with the results of clinical findings with several chickens, some of 

 which were affected with fowl diphtheria and fowl cholera. The author was 

 not able to verify Ellermann and Bang's findings that the normal ratio of white 

 to red corpuscles in the fowl is 1 : 100. With normal healthy hens it was 1 : 250 

 and with pigeons 1 : 300. Further, it is concluded that the leucocytosis observed 

 by these authors must be attributed to another cause than the infective agent of 

 leukemia. 



