584 EXPERIMENT STATION RECORD. 



found only in chicks under 6 weelcs of age it is evidently quite common in the 

 State. The course of the disease is quite rapid with a mortality of from 30 to 

 50 per cent of the chicles hatched. The report given is based upon an exami- 

 nation of 17 chicks. The author describes the sj^mptoms and post-mortem 

 appearances, and gives a complete account of a typical case. The causative 

 organism is thought to be closely related to if not identical with the organism 

 described by Smith (E. S. R., 7, p. 524), as Amaha meleagrhlis. Remedies pre- 

 viously suggested by the Bureau of Animal Industry of this Department (E. 

 S. R., 19, p. 988) are discussed. 



A bacterial disease was discovered in a flock where it was very fatal to 

 chicks under 5 or 6 weeks of age. The course of the disease is said to have 

 been very rapid, chicks dying during the night without previously having 

 shown signs of illness. The symptoms and results of post-mortem and micro- 

 scopic examinations are reported accompanied by a description of the organism. 

 Two chicks and one of three mice died when inoculated with cultures of the 

 organism. 



The action of the aggressin of fowl cholera, E. Weil (Arch. Hyg., 65 (J90S), 

 No. 2, pp. 81-106). — The aggressin isolated by the author from cases of fowl 

 cholera produces a striking susceptibility when inoculated into fowls. Control 

 birds resisted a dose of virus ten times as great as was required to kill fowls 

 previously treated with the aggressin. The aggressin, however, seems not to 

 possess any toxic properties, since no injurious effects were produced in the 

 experimental animals by increasing the dose eight times. A brief bibliography 

 relating to this subject is appended to the article. 



Fowl plague with special reference to its pathological anatomy, Freese 

 (Dcut. rier(ir::tl. WchH.uhr., 16 (1908), No. 12, pp. 173-177}.— Yowl plague is 

 known to affect chickens, turkeys, guinea fowl, pheasants, geese, and sparrows. 

 The lesions of the disease resemble those produced by poisoning with phos- 

 phorus. It is to be distinguished from fowl cholera by the presence of hemor- 

 rhages under the epicardium. an exudate in the pericardium, and intense 

 enteritis in the case of the latter disease. Until recently no good evidence has 

 been obtained in Germany that this disease affects geese. A number of cases 

 of si)ontaneous infection were noted. 



Studies and micro-photographs of the pathogenic protozoa, T. von 

 VVasielewski (Stiidien und Mikropltotograiinnc zur Kcimtnia dvr Patliogcnoi 

 Protozoal. Leipsic, 1908, pt. 2, pp. 175, pis. 8, figs. 26). — A detailed account is 

 given of the distribution of animal parasites in the blood in Germany. The 

 author had occasion to study these parasites in material obtained from birds 

 and mammals. Particular attention is given to Filaria, trypanosomes, hema- 

 gragariues, Plasmodium, especially the species found in birds, Hemoproteus, 

 and Leucocytozoon. The blood parasites which cause plasmodiosis of birds 

 were followed through 45 generations. 



Animal parasites, E. Perroncito {Ana. R. Accad. Agr. Torino, ^9 (1906), 

 pp. 3-17). — Par;!sitism of young pigs with Rhabdoncnia istrongyloidvs is some- 

 times so excessive as to cause death. Notes are given on fatal infestation with 

 these parasites. The lungs, heart, pleura, and peritoneum showed no lesions. 

 Brief notes are given on sanitary methods of preventing reinfestation with these 

 parasites. 



A study was made of multilocular echiuococci in sheep. These parasites ap- 

 pear to be very common in sheep according to the author's experience. A form 

 of cachexia is described caused by fluke worms and accompanied with jaundice. 

 In cases of excessive infestation it is held that anemic and cachectic conditions 

 often develop. 



