1020 . EXPERIMENT STATION KKCORD. 



in (liffeivnt years from 1888 to 1902. The syniptoiuH are fever, reddening of the 

 nHicons nienil)raneH, discharge.s from tlie eyes, swelling of the eyelids, swelling of the 

 legs and in the skin and other parts of tlie Ixxly. The disease attacks young horsea 

 more frequently. Success was liad in treating the disease by proper attention to diet 

 and ])y rubbing affected parts with spirits of camphor and antiseptic solutions. 



The treatment of morbus maculosus with Ichthargan, Lanok (//.sc//r. 

 Vderhiark., 1,5 [190.3), Xo. S, pp. 117-119). — For horses of me<lium size Ichthargan 

 was found to produce good results when injected in doses of \^ gm. per day. Experi- 

 ments showed, however, that this remedy does not exercise any effect u))on the 

 organism of morl)us maculosus except in the circulating blood. Tt is recommended, 

 therefore, that the treatment l)e repeated daily. 



A skin eruption on the head of horses, ScnEFKiu.iNd {Ztschr. VHcr'niark., 1.5 

 {190:1), Xo. 7, pp. :l.^^J-,S^6, Ji(j. l). — This disease occurred in a nundier of horses and 

 appeared to be confined entirely to the head. It began as a rule at the corners of 

 the mouth and progressed backwards to the ears. Pustules were formed and the 

 hair of affected parts was shed. The lymphatic glands of affected regions were also 

 swollen and became distinctly visible. These symptoms could ]>e controlled, how- 

 ever, by external treatment with potassium iodid. In some of the pustules Stuplty- 

 lococcus pyogenes aureus were found. Inoculation experiments with this organism, 

 however, failed to reproduce the disease in any case. The true cause of the disease 

 was believed to be a mite, Clwi/letus rrnditns, which was found in the skin and in the 

 Rivollen lymphatic glands. 



Lymphangitis, Coi>l.\ri) {Eec. Med. Vet., S. .ser., 10 {1903), No. 22, pp. 502-504).— 

 This disease, which is known in many parts of France and elsewhere under the name 

 edema, appears suddenly and leads to the development of high fever in infected 

 horses. The etiology of the disease appears to be obscure and so far as experiments 

 have been made treatment is unsatisfactory. 



Apoplectic hemorrhagic septicemia of nevr-born colts, C. Darmagn.\c {Eec. 

 Mt'd. Y('i., S. ser., 10 {190.3), Xu. 11, pp. 37S-.377). — Detailed clinical notes are given 

 on a number of cases of this disease. From a study of these cases the author comes 

 to the conclusion that infection takes place through the umbilical ves.sels, and that 

 the sudden development of the disease is due to the rapid spread of the toxin l)y 

 means of the circulation. Good results have been obtained in controlling the disease 

 from the application of thorough antisepsis to the umbilical cord, as recommended 

 by Nocard in combating white scours. 



The occurence of ticks on horses, D. .Tuxack [Ztscln-. Yeterinarl:, 15 {1903), 

 No. 6, jyp. 258, 259). — Brief notes are given on the excessive infestation f)f horses Ijy 

 Ixodes ricinus. These ticks fasten themselves to the skin of horses in almost any 

 location, and their occurrence in unusually large numbers caused the falling of the 

 hair on infested areas and also other intlanimatory processes in the skin. 



Colics of the horse and their treatment, Y. Dkouin {Rev. Gm. Med. Vet., 2 

 {1903), No. 23, pp. 569-5S2). — The various forms of colic which have been observed 

 in the horse are classified according to their etiology and symptoms. Notes are given 

 on the use of asafetida, opiates, Indian hemp, carbonate of ammonia, nux vomica, 

 eserin, barium chlorid, and other drugs in the treatment of colics. 



A pathology for forage poisoning, or the so-called epizootic cerebro-spinal 

 meningitis of horses, I). .1. ^IcCartiiy and ^I. P. Kavexkl {.lour. Jfed. Resinrclt, 10 

 {1903), No. 2, pp. 243-249, pi. 1) . — In certain cases of so-called epizootic cerebro- 

 sjjinal meningitis the cause was clearly traced to the feed which the horses had eaten. 

 While feeding experiments conducted in connection with a spontaneous outbreak of 

 this trouble showed that the disease was due to spoiled feeding materials, it was not 

 possible to isolate any pathogenic organism which caused the trouble. 



A careful post-mortem examination of animals affected with this disease revealed 

 the presence of lesions in the anterior i)ortion of the alimentary tract and also in 



