MICROSPORIDIA , HELICOSPORIDIA 



319 



each of which produces a number of spores characteristic of each 

 genus. Some spores seem to be capable of germinating in the 

 same host body, and thus the number of infected cells increases. 

 When heavily infected, the host animal dies as a result of the de- 

 generation of enormous numbers of cells thus attacked. Such 

 fatal infections may occur in an epidemic form, as is well known 

 in the case of the pebrine disease of silkworms, the nosema- 

 disease of honey bees, microsporidiosis of mosquito larvae, etc. 

 According to the scheme of Leger and Hesse, the Micro- 

 sporidia are divided into two suborders as follows: 



The spore with a single polar filament Suborder 1 Monocnidea 



The spore with two polar filaments Suborder 2 Dicnidea 



Fig. 137 a, b. Nosema bomhycis. (After Kudo), a, a fresh spore 

 (XllOO); b, a silk- worm larva showing typical 

 symptoms of heavy infection (one-half natural 

 size), 

 c, d. N. bryozoides. c, portion of infected faniculus cut 

 longitudinally (X200 after Braem); d, a stained 

 spore (X900 after Schroder). 



e. Four fresh spores and one stained spore of A'', apis. 

 X1170 (After Kudo). 



f. Four spores of N. cyclopis. X1170 (After Kudo). 



g. Two spores of N. anophelis. XI 200 (After Kudo), 

 h, i. Glugea anomala. h, cross-section of Gasterosteus 



aculeatus infected by the microsporidian (after 

 Thelohan); a spore (X1125 after Stempell). 

 j. Stained spore of G. hertwigi. X1250 (After Weis- 



senberg). 

 k. Two spores of Perezia mesnili. X600 (After Pail- 

 lot). 

 1, m. A pansporoblast and a spore with its extruded polar 

 filament of Gurleya richardi. X900 (After Cepede). 



