1342 Journal of Applied Microscopy 



A destruction of the muscle does not always take place. The author de- 

 scribes a primary interstitial non-purulent form of myocarditis in which collec- 

 tions of cells pushed aside the intact muscle-fibers. He regards this variety 

 as toxic in origin. The foci of cells are at length replaced by fibrous nodules. 



The seat of the disease in the blood-vessels can be in the main branches of the 

 coronary arteries, or outside the heart in the root of the aorta, or in the orifices 

 of the coronary arteries. 



Fujinami concludes that fibrous myocarditis originates in a variety of ways. 

 It is simply the final outcome of a number of different pathological processes. 

 The thickenings of the vessel-walls, demonstrable microscopically, are not al- 

 ways to be regarded as the cause of the formation of the fibrous areas. The 

 vascular changes can occur as a result of the fibrous myocarditis just as the 

 vessels in the scar tissue of healing ulcers become sclerosed. 



Fragmentatio myocardii is frequently associated with sclerosis of the coron- 

 ary arteries and fibrous myocarditis. j. h. p. 



„, .. . . . , , T, , ■ , According to Schmorl, accessory ad- 



Wartnin. Accessory Adrenal Body in the ° ■' 



Broad Ligament (Adrenal of Marchand). renals are found in the neighborhood 

 American Journal of Obstetrics, 42 : 797- ^f ^j^g adrenals, in the adrenal and 

 805, 1900. 



solar plexuses, and along the adrenal 



and spermatic veins, in 92 per cent, of all autopsy cases. Accessory adrenal 

 tissue has been found in the kidney capsule and cortex. Along the spermatic 

 vein, in the pampiniform plexus, between the testis and epididymis, in the cor- 

 pus Highmori, pancreas, liver, and broad ligament. 



The author was able to collect from the literature only 23 cases of accessory 

 adrenals in the broad ligament. Marchand in 1883 was the first to report this 

 anomaly. The diagnosis is made by the characteristic epithelial-like cells, and 

 the relation of these cells to the connective tissue and capillaries. Usually the 

 structure of the body is uniform throughout, in some cases resembling the cortex 

 and in others the medullary portion. As a rule the accessory adrenals of the 

 broad ligament consist of cortical tissue only. The accessory adrenal found by 

 Warthin was a pale yellow, fat-like body of the size of a pea. It was situated 

 behind the ovary, near the plexus of veins. j. h. p. 



Abbott, M. E. Pigmentation Cirrhosis of the An advanced cirrhosis of the liver and 

 Liver in a Case" of Hasmochromatosis. , , ^ 1 • • 



Journal of Pathology,?: 55-69, 1900. ^ moderate degree of chronic mtersti- 



tial pancreatitis were associated with 

 an extensive deposit of iron-containing pigment in the tissues. There was a 

 bluish gray slaty tinge of the skin, and a rusty brown discoloration of the inter- 

 nal organs. Sections of the liver and pancreas were loaded with golden-brown 

 pigment, responding with a deep blue color to Perl's test for iron, which was 

 present also, though in a lesser degree, in the spleen, suprarenals, and heart 

 muscle. There was more or less fibrosis of all the organs except the kidney. 

 In both liver and pancreas the heavy pigmentation of the connective tissue had 

 its source, in part at least, in the broken-down pigmented cells of the paren- 

 chyma. A fairly advanced chronic interstitial pancreatitis existed without the 

 clinical picture of diabetes so common in cases of advanced ha^mochromatosis. 



