SPIROCHETOSIS OF SUDANESE FOWLS 79 



demonstrated. If the hen-house is infected, he says that infection per as can 

 take place. 



17. Tedeschi^ by the use of abrin killed Sp. (laUinai-niu, and thus produced a vaccine -V pioteciive 

 whicli protected canary birds. It is worth noting that he found that iS'^. duttoni 



in experimental animals during fever-free periods, remained in the blood as endo- 

 globular forms, both in the erj-throcytes and the leucocytes. 



18. Dodd- has a paper on fowl spirochaetosis in Queensland. He describes the symptoms 

 well, but there is not much that is new in his account. He did not find the 

 "after phase" bodies despite careful search. 



19. Blaizot'^ contributed a paper on relapses in the fowl spirochaetosis of Tunisia 

 (virus de Degache). He cites the work of Conite and Bouquet, already mentioned, 

 and also the facts as regards recurrence discovered by Bouet and myself. The object 

 of his research was to discover if relapses, characterised by the presence of spirocho'tes 

 in the blood, occurred in the Tunisian spirochetosis. 



This he found to be the case and he points out that so far as we know at 

 present only a heavy, acute infection is capable of conferring a permanent immunity. 

 He points out the close relationship existing in this direction between African fowl 

 spirochaetosis and the spirochetal fevers of mammals. He puts forward the ingenious 

 suggestion that both relapses and absence of immunity may be due to a premature 

 crisis occurring before there has been time for saturation with auti-bodies and thereby 

 preventing the first infection being sutificiently severe to ensure a complete immunity. 

 From the study of this question of inmiunity Blaizot believes that it may yet be 

 possible to affirm that Africa is the original country whence avian spiroeh»tosis spread. 

 His paper is interesting, suggestive and will repay careful perusal. 



20. Quite recently, Jowett^ has discovered fowl spirochaetosis in the vicinity of 

 Cape Town. Moreover, along with the disease, he has found intra-corpuscular 

 bodies exactly like those in Sudan fowls. A. pcrsicus is the vector, and, judging 

 from the preliminary paper, this spirochaetosis of South Africa is identical with our 

 Khartoum form. 



21. Gilruth'J describes the disease from Victoria, in Australia, but did uot find 

 endoglobular forms. 



22. Dschunkowsky" has recently treated the spirillosis of geese successfully with "606," Successful use 

 which he finds to be much more active than atoxyl. 



23. Blaizot" in yet another paper returns to the subject and deals with the subsequent 

 cachexia, the question of intracerebral inoculation and the susceptibility of fowls to 

 second infections. 



• Tedeschi, A. (1910), " E.xpenraenteUer Beitrag zur Recurrensfiebers (Spirochcele Duttoni)." Cent. f. Bakl. 

 Oriij., Abt. I. Bd. 54, H. 1, p. 12. 



-Dodd, S. (March 31, 1910), "Spirochaetosis in Fowls in Queensland." Journal of Comparatire 

 I'litholuyi/ anil Therupeulics, Vol. XXIII., part 1. 



■' Blaizot, L. (1910), "Note sur la recurrence dans la Spirochsetose des Poulcs en Tunisie (virus de 

 Degache)." Arch, de VInst. Past, de Tunis, part 2, p. 53. 



* Jowett, W. (December, 1910), "Fowl Spirochsetosis, Note on the Occurrence of, at the Cape." — 

 T/ic Atji-ie.uUnnd Juarnal of the Cape nf Good Hope, Vol. XXXVII., No. 6. 



■'■ Gilruth, J. A. (1910), "Note on the existence of Spirochaetosis affecting Fowls in Victoria." Proceedimjs 

 lioj/al Sociclii, I^ietoria, Vol. XXIII. 



» Dschunkowsky, E. (.January 5, 1911), " Heilversuche rait Ehrlich-Hata " 606 " bei der Qansespirillose, etc." 

 £cr!. ticrarzlh Il'och. 



■ Blaizot, L. (1910), "Nouvelles Recherches sur la Spirochetose des Ponies." Arc/i. de rin.tt. Past., Tunis, 

 part IV. 



