Emigration in Passive Hyperemia. 37 



try of the world. These figures arranged in order of magnitude 

 nearly represent commercial value for textile fabrics, but for 

 paper stock other conditions modify the result. 



EMIGRATION IN PASSIVE HYPEREMIA. 



BY W. T. BELFIELD, M. D., OF CHICAGO.* 

 {Received Aug. lUk, 1878.) 

 In March last I had occasion to superintend the post mortefn 

 examination of a case of pneumonia, in which death had oc- 

 curred on the tenth day of the disease. There was found con- 

 solidation of the entire left lung, gray hepatization in the lower 

 lobe, red in the upper. Nothing peculiar was noticed in the 

 other organs except general engorgement, especially marked in 

 the kidneys. There had been during our observation of the 

 patient not a solitary symptom of renal disease except the 

 presence of albumen (about 5^,) in the urine — a presence 

 known to be common in pneumonia, and attributed to me- 

 chanical congestion. For at least three features of pneumonia 

 tend to the production of mechanical congestion, namely: 

 the decrease in breathing surface, the increase in the demand 

 for oxygen made by the excessive tissue change, and the fee- 

 bleness of the heart's contractions. Hence the albuminuria 

 and the post ?nortem engorgement of the kidneys were regarded 

 as legitimate results of the disease. A happy curiosity, how- 

 ever, led me to make a microscopical section of the kidney. I 

 found the tubules rather smaller than usual, the intertubular 

 spaces and capsules of the malpighian tufts much thickened by 

 the presence within them of numerous small, round, finely 

 granular cells ^^^^ to sq'ot i^ch in diameter. These cells had 

 every appearance of white blood corpuscles, and were so pro- 

 nounced by Dr. Danforth and others. By way of explanation 

 it was presumed that the retardation of the blood current, due 

 to the causes previously mentioned, had afforded the colorless 

 corpuscles an opportunity to exhibit their amoeboid movements 

 — and that the opportunity had been improved. That this 

 was not an inflammatory process, was proved by the absence 

 from the clinical history of all the recognized symptoms of 

 renal inflammation, and by the absence from the urine of the 



* Read before the National Microscopical Congress at Indianapolis, August i6th, 1878. 



