xviii Journal of Comparative Neurology. 



months before the glands of the neck were removed on account of 

 tubercular disease. Complete unilateral palsy of the right side of the 

 tongue, soft palate, pharynx, and right recurrent laryngeal nerve 

 developed. Autopsy showed the nidulus of the 12th nerve greatly 

 altered on the right, and less so on the left. Niduli of the ninth, 

 tenth and eleventh nerves were somewhat affected. The respiratory 

 bundle appeared completely degenerated on the right side, while on 

 the left, in the region of the hypoglossus nidulus its ventral and ceph- 

 alic parts were diseased. No evidence of tubercular disease was found 

 and the change was considered motor atrophy. 



Dr. Wiener summarizes as follows : 



1. The region of the hypoglossus nucleus gives origin to nerve 

 fibres which supply the tongue, palate, pharynx and larynx on one 

 side of the body. 



The column of nerve fibres known as the respiratory bundle con- 

 sists of fibres from the glossopharyngeal, vagus and vago-accessorius 

 fibres. 



3. The glossopharyngeal nerve seems to control the reflexes of 

 nausea and gagging in the soft palate and pharynx, and also to send 

 some of the motor filaments to the pharyngeal muscles. These latter 

 filaments take their origin in the hypoglossal nucleus and ascend in the 

 respiratory column to the nucleus proper and then make their exit 

 with the glosso-pharyngeal nerve. 



4. The soft palate muscles are not innervated by fibres from the 

 seventh nerve. {Jonrn. Nerv. Mental Diseases, Jan. 1894.) This 

 case may be compared with that recorded by Reinhold (Deutsch. 

 Zeitsch. f. Nervenheilk. Oct. 1893.) which had been reported as one of 

 bulbar paralysis. After 12 years, during which the symptoms had 

 not changed, the patient died of bronchitis and microscopic examina- 

 tion showed no structural changes. The case proved to involve prim- 

 ary disease of the muscles. "Dystrophia muscularis progressiva." 



In his paper on Hereditary Spastic Spinal Paralysis {Deutsch. 

 Zeitsch. f. Nervenheilk, Oct., 1893.) Striimpell states that: 



1. Under the influence of an abnormal hereditary defect there 

 develops, slowly but progressively, a primary systematic degeneration 

 of the crossed pyramidal tracts. 



2. The symptoms (primarily sharply limited spastic disturbance 

 of the lower extremities) develop between 30 and 40 years of age. 



3. After the lapse of years the disease generally becomes a true 

 spastic paresis and paraplegia of the legs. 



4. As a rule, the pathological changes limit themselves to a de- 



