1142 
discover this deviation of the «-v-interval directly after the larger 
pauses for the poly- and trigeminusgroups. The reason of this pheno- 
menon is. that here the ventricle-systole begins during the systole of 
the preceding auricle (vide Figs. 1, 2, 3, and 4). Consequently these 
two shortenings sum up and the systole of the ventricle is not 
retarded in the curves. From this it appears again, that the repair 
of conduct in the connection-systems becomes constantly worse after 
the falling out of one systole. Till the trigeminus-groups (these included) 
the first ventricle-curve after a hiatus begins during the systole of 
the preceding auricle; with the bigeminus-groups the first ventricle- 
systole begins during the diastole of the preceding auricle-curve. 
(I can explain with another example the fact, that the curve of the 
systole of one partition of the heart changes entirely, when at the 
same time another partition of the heart is in diastole (vide Fig. 5). 
The third row of curves is here represented 45 min. after the 
injection of 5 drops of 1°/, acetas veratrini. Here the auricle-systole 
begins at the 3°¢ and 5" curves in the beginning of the diastole of 
the ventricle, and then, as it were, it sinks entirely away; if, on 
the contrary, the auricle-systele begins at the top, then it is much 
larger. This piece has been photographed from a row of 2'/, m. in 
length, in which, whenever the auricle occurs in the BE of 
the ventricle-diastole, the latter decreases. In the following row of 
curves the rhythm is halved, and an everywhere equal auricle- 
svstole is represented on the ventricle-curve. ) 
| have thus noted for 1°/, hour bigeminus-groups of this heart; 
Fig. 6. 
Transition from pbigeminus into the normal rhythm, then again 
p. bigeminus and afterwards halving of rhythm. 
