( 1199 ) 
D. Disease of Stokes-Adam (Wrpacw’s armlet). 
MBp + Aw. 19% 
mBp + Aw. 80 
Pulse amplitude Lif 
A few determinations of the arterial wall were too great because 
the determination was made too quickly. Not taking into account 
however these determinations we were struck by the fact, that the 
factor found for the arterial wall with the same person, is rather 
constant, even with different MB. This proves of course strongly 
in favour of the method. 
Only onee in the 172 determinations the determination of the 
MBp + Aw is doubtful, most likely on account of the influence of 
the expansion of the air in the plethysmograph by warmth. All 
other determinations could be read with perfect accuracy. 
The determination of the MBp without the Aw requires greater 
care and to state this we may not be satisfied with a few deter- 
minations. 
With normal persons as a rule the J/4p increases with their ages 
and so does the mAp, which was not unknown. 
The Aw factor does not vary in the same degree. On an average 
it is 6—10 mm. Hg. Consequently the mistake is not great, if we 
consider for normal people the MBp + Aw determined according 
to the obliteration-method as the maximal blood-pressure. And with 
our method the latter can be quickly and easily determined, whilst 
the determination of the Aw requires time, and is somewhat painful 
with high blood-pressure. But it is only so with high blood-pressure. 
One of our patients with a maximal blood-pressure of 135 mm. He. 
did not feel any pain, another with a maximal blood-pressure of 
170 mm. Hg. did feel pain. 
In general the figures found by us do not deviate from those 
which are usually admitted as normal. 
It is remarkable that the influence of the Aw that was found with 
arteriosclerosis was very slight. Perhaps we may not conclude too 
much from our single case. Yet a slight influence of the Aw with 
strongly pronounced arteriosclerosis would perhaps not be fully in- 
explicable. In such an arterial wall we find besides calcified parts 
which undoubtedly offer a greater resistance to the compression than 
a normal wall, certainly also degenerated parts of the nature of a 
fibrous tissue, the elasticity and resistance of which have diminished. 
This view seems to be confirmed by the decrease — though only 
in a slight degree — of the Aw with the age. 
