828 



the degeneration of the nerve was not equally divided through the 

 whole breadth of the nerve, there was sufficient proof that the nerve 

 had l)eeii submitted directly or indirectly to a noxious iutlueuce. 

 Therefore it was necessary to pursue the exaniiuatiou up to the 

 spinal ganglion. It then apfieared that the whole proximal part of 

 the nerve was degenerated and the nervefibres in the spiualganglion 

 as well were swollen and rich in lipoid lumps. 



The iiervecells in the ganglion had altered and were surrounded 

 by phagocytes. Here and there „Babes Kuötciien" (an accumulation 

 of small cells in the midst of which were lying some skeletons of 

 nervecells) were visible. (Fig. 11). 



Pursuing the radices up to the medulla, we got nearly the same 

 image of a degenerated iiei-ve as was shown to us by the more 

 peripherical parts of the nei've. The swollen cylindre axis is l^'iiig 

 in a hollow tube from which the myelin has totally (lisap|ieared 

 (Fig. III). This degeneration of the roots could be follo\\ed into 

 the white matter of the medulla spinalis, allliougli between the 

 degenerated tubes there were always fibres (piite noiinally built. 

 The uei'vecells in the segment of the metlulla coi-iesponding with 

 the examined nerve had partly lost their N/ss/ lumps, partly were 

 they recognizable only by a pale nucleus, surrounded by a mass 

 of small gliacells (Fig. IV) JS^ttgr ibodiefi were not found here. 



Fig. III. 

 Lumbal root of the inoculated 

 side with degeneration and 

 loss of the myelin sheatlis. 



Fig. IV. 

 Neuronophagy. 



Microscopical slices impregnated with silver showed that the 

 intracellular fibrils either had disappeared or had doited together 

 to big threads {Golgi alterations lig. V). 



Just as it was found in the s|)inal ganglion the degenerated cells 

 lay scattered among quite luirmal cells, so that the examination 



