It would tako me too long- in this |)aper to enter inoie in 

 particular into this hypothesis; I refer the interested reader to the 

 original connnnnicalion. The one thing- to which 1 will draw atten- 

 tion, is, that according to this view, considering oni- etpiilihrinin, 

 there shonid he made no principal difFerence hetwoen tiie afïerént- 

 proprioceplive stimuli which are conducted from the cerebellai" tracts 

 of the lateral colninn and those from the veslihnlai- apparatus to 

 the cerebellum. All these stimuli are related to the e(piilil)rium- 

 sensation and therefore regulate our gait. These different alfercnl 

 tracts thus form a whole and thej are to he considered as a sub- 

 division of the same equilibrium system. 



Now it is important to trace, whether they change, and if so, 

 what alterations these afferent stimuli undergo, when thev arrive 

 in the cerebellum. 



If, investigating this, one makes patients, suflering from e.g. 

 cerebellar tumors, perform the same walking-tests, as I did with 

 suiïerers from tabes or vestibular affliction, then the residts prove 

 to be totally the same. 



For instance I will report the following case, in which dui-ing 

 life the diagnosis was made of a tumor, which was located at the 

 left side, where it pressed as well on the cerebellum as on the 

 nervus octavus. During the operation and also post-mortem the 

 diagnosis could be confirmed. 



If one had the patient walk totally unsupjiorted with Often eyes, 

 then the part of her gait was obtained as reproduced in fig. 1. The 

 ataxia is distinctly visible. If her eyes were shut, then the path of 

 her gait became as in fig, 2. 



When comparing the two the ataxia proves to be considerably 

 augmented. This is comprehensible, because in the latter case the 

 afierent-extero-ceptive stimuli from the eyes are missing. The differ- 

 ence between these two paths of gait results from the influence of 

 the eyes on the performed movements. 



It is important to point out once more (see preceding paper) that, 

 when by abnormalities of the static organ a second organ comes to 

 his aid, the latter only can partially leplace the deficiencies of (he 

 former, if these are of rather severe nature. 



This is distinctly visible in fig. I, because notwithstanding the 

 aid of the eyes, the ataxia however is far from gone. 



If we now let such a patient walk quite free with eyes open, 

 but giving her contact sensation through persons next to her, then 

 althongh also the, neck-equilibrlnia-iinpiilses (we shut out by means 

 of a bandage, the gait path in fig. 3 is reproduced, in which the 



