PART III. TREATMENT 



TABLE X. continued. 

 (13) Acute Cases sJiawiiig Anicehe loith included Red Blood Corpuscles. 



101 



1 When course of emetin injections was completed patient was given emetin grain by the mouth 

 for seven days. 



Was given later a twelve-day coursa of emetin one grain a day by the mouth to try and get rid of 

 the Entamcebce coli infection. 



* Had a grain of emetin by the mouth on the two nights before the injection course was started. 

 J Had relapsed after eight injections of emetin three weeks before. 



" Cured later by emetin one grain injection with grain by the mouth each day for twelve days. 

 B Not treated further. 



7 Cured by course of emetin by mouth given immediately afterwards (5 grains). 



8 Cured later by courses of one grain of emetin by mouth for twelve days. 

 '' Relapsed again later after course of emetin by mouth with injections. 



10 Had relapses after emetin grain a day for twelve days. 



11 Had resisted courses of emetin by mouth one grain a day for twelve days. 



12 Afterwards found resistant to 1 grains of emetin by mouth for twelve days, ipecac, and thymol. 



A = Degree or size of infection. B = Result. C = Number of days after commencement of 

 treatment before infection disappeared. D = Number of days of control before cure or relapse was 

 noted. F = History of previous dysentery. G = History of previous emetin treatment. CR = Cure. 

 R = Relapse. NR = No reaction. 



Two of the cases, Healy and Spiers, in Group I, were passing 

 only cysts of E. histolytica and amoebae without included red blood 

 corpuscles when first seen, but both of these cases were just recover- 

 ing from attacks of acute amoebic dysentery which they had had 

 repeatedly for some years. They were really in a transition stage 

 between the condition of the carrier and acute amosbic dysentery. 

 In this group there were fifty-eight cases, of which six were acute 

 cases of amoebic dysentery. Of the 52 carrier cases 37 were cured 

 (CR), and did not relapse in the period of control, 10 relapsed 

 (R), while in 5 the infection did not even disappear, there being 

 apparently no reaction to the treatment (NR). The six acute' 

 cases all relapsed. It is interesting to note that of the carriers 

 who relapsed only one gave a previous history of dysentery, while 

 three of the five cases which did not react had had previous 

 dysentery. Of the thirty-seven carriers which were cured fifteen 



