170 MANUAL OF POISONOUS PLANTS 



likewise probable therefore, that it is due in large part to the opsonin content 

 of the blood. There are no bactericidal or anti-toxic substances formed in 

 immune blood. The blood serum of an immunized individual exhibits an in- 

 creased agglutinating power. Immune sera for the prevention of the disease 

 have not proven a success. 



^ 



Micrococcus tctragcnus, Gaffky 



Disease produced. Associated with the tubercle-bacillus, and probably of 

 importance in complicating the pus infections. 



Animals infected. Man and animals. 



Animals susceptible. White mouse, and guinea-pig. 



Animals immune. House-mouse, field mouse, dog, and rabbit. 



Pathogenesis. Inoculation of white mouse causes fatal bacteremia. The 

 organism is found in tubercular infections, and probably hastens the necrosis 

 of infected tissue. In some cases it may be the primary infecting agent. 



Poisonous properties. Not known. 



Immunity. Not known. 



Micrococcus catarrhallis, Seifert 



Disease produced. Superficial inflammations of the respiratory tract and 

 conjunctiva. 



Animals infected. Man. 



Animals susceptible. None of the laboratory animals are susceptible, except 

 when the organism is introduced in very large quantities. 



Pathogenesis. Probably the primary cause in some cases of conjunctivitis,, 

 bronchitis, and catarrh, and in general superficial inflammations of the respira- 

 tory passages. 



Poisonous properties. Not known. 



Immunity. Not known. 



Micrococcus gonorrheac, (Diplococcus of Neisser~) 



Disease produced. Gonorrhea. 



Animals infected. Man. 



Animals susceptible. None of the laboratory animals are susceptible. 



Pathogenesis. Producing a severe inflammation of the mucous membranes 

 of the urethra accompanied by blennorrhea. Secondary infection of fallopian 

 tubes, ovaries, urethra, etc., may occur. 



Poisonous properties. Not known. 



Immunity. Probably some immunity is developed after infection, but is 

 not lasting. No method of immunizing is known. 



Streptococcus equi, Schiitz 



Disease produced. Strangles. 



Animals susceptible. Horses, asses, and their hybrids, and mouse. 



Pathogenesis. Producing a severe catarrh of the nasal mucosa, with a 

 swelling of the sub-maxillary, and pharyngial lymphatic glands, abscesses gen- 

 erally form in the latter. May terminate in pharyngitis in a purulent pneu- 

 monia or pleuritis. Sometimes cutaneous exanthemata. Metastatic abscesses 

 may appear in various lymph glands. 



