112 PERI-HEPATITIC ATROPHY CHARACTERISTICS. 



These granulations, however, are formed in quite a 

 different manner viz., the roots of the hepatic veins 

 are enlarged as far as their capillary origin, and cause the 

 surrounding cells to disappear. In this way the ] 

 corresponding to the hepatic veins sink down, whilst 

 those occupied by the portal vein project in tine granu- 

 lations. At first the wasting of the organ is confined to 

 the circumference of the enlarged capillaries, but, after a 

 time it extends to the large branches, and as a conse- 

 quence produces extensive depressions, and new con- 

 nective tissue is developed around the vessels, impart in- 

 to the organ a certain amount of firmness. This form of 

 atrophy of the liver is frequently hypertrophy 



ami valvular disease of the heart. 



3. rvLi:-riiLKr,iTic ATUOPHY. A wa^tin^ of the, U 

 will sometimes follow an obliteration of the line brandies 

 of the portal vein the result of "adhesive phlel< 

 On tracing the branches of the portal vein into the 

 interior of the gland, we arrive at blind extrein 

 which are sun-minded by a linn shrivelled tissue, the 

 remaining portion of parenchyma bring left unchai 

 depressions are thus formed, \\hidi uive to the liver a 

 lobulated, and at other times a coarse granulated ap] 

 ance not unlike that of cirrhosis. This condition however 

 differs materially from the latter disease viz., b\ 

 greater size, its less regular form, the flatter character of 

 its projections, by the total absence of the bands of 

 connective tissue in its interior, and by there being no 

 history of SPIRIT DKINKIV 



1 rr.Ki-HKPATiTic ATROPHY. There is a form of 

 wasting of the liver which follows repeated attacks of 

 inflammation or its capsule (peri-hepatitis). In such 



