36 THE ANIMAL PARASITES OF MAX 



apparently, the penetration is not so great as in men, according to Kruse and 

 Pasquale. During their migration the parasites also gain access to the lymph- 

 follicles of the wall of the intestine, which become swollen and commence 

 to suppurate ; follicular abscesses arise and after their rupture follicular ulcers. 

 The diseased patches in the mucosa are markedly hyperaemic and numerous haemor- 

 rhages are set up. Roos and Harris state that the amcebae also oenetrate into 

 the blood-vessels (fig. 7) and this explains the occurrence of metastatic abscesses. 1 

 The whole submucosa is severely swollen at the diseased spot and undergoes small- 

 celled infiltration in the neighbourhood of the colonies of amcebae. From these 

 findings Jiirgens (1902) draws the conclusion 2 which is followed here, that the amcebae 



FIG. 7. Section through wall of large intestine (of a man) close under an ulcer caused by 

 Entamcfba histolytica. A, amoebae that have penetrated partly in blood-vessels (Bv), partly 

 in tissue of submucosa to the muscularis. Magnified. (After Harris.) 



are causative agents of the enteritis of cats, which disease is well defined, both 

 pathologically and anatomically. Subsequent researches confirm the experience of 

 earlier authors ; great precautions were taken to exclude errors, hence, as with 

 Gross and Harris, no exception can be taken to their results. The inoculation 

 material was derived from soldiers who suffered from amoebic enteritis in China 

 and who were admitted into the garrison hospital at Berlin. In order to be 



1 Lung abscesses generally arise by the bursting of a liver abscess through the diaphragm 

 into the right lower lobe of the lung, sometimes also through conveyance of amosbse by means 

 of the blood-stream (Banting). 



2 These findings were confirmed by Schaudinn by means of investigations on cats and men. 

 Cf. also Alfred Gross, Marchoux, P. G. Woolley, W. E. Musgrave, H. F. Harris and others. 



