262 [Recess, 



although it facilitates its production. In corrohoration of these views 

 I may refer to the well-known results following the section of various 

 nerves, e. g. the pneumogastric and the fifth. 



This muscular paralysis is probably producible directly by muscle- 

 sedatives, as it is indirectly by muscle-irritants. Since diminished 

 nerve-force produces hypersemia, and since diminished nerve-force 

 furnishes the conditions under which homogeneous, i. e. inflamma- 

 tory, stasis is most prone to occur, we see why hypersemia and 

 inflammation are so frequently conjoined. The experiments of 

 Claude Bernard on the sympathetic, while showing the connexion 

 between neural paralysis and hyperaemia, also indicate that neither 

 neural paralysis nor hypereemia are convertible terms with inflam- 

 mation. 



A distinction is drawn between that diosmotic exudation which 

 leads to homogeneous stasis, and that subsequent copious transudation 

 of fluid which fills up the interstices of tissues or leaks into cavities J 



If in a frog's web homogeneous stasis has occurred in the venous 

 radicles so as to completely prevent the passage of the blood into the 

 veins, the current in the capillaries and supplying arteries might 

 naturally be expected to be brought to a stand, as it certainly would 

 be if the walls of the capillaries removed from the immediate seat of 

 the obstruction were impervious ; but so far from this being the case, 

 the blood brought to the part is seen to pass on in a perfectly regular 

 manner without the slightest rebound. This absence of rebound is 

 an evidence that the liquor sanguinis is passing through the vascular 

 parietes at the same rate it is being propelled into the obstructed 

 vessels. 



It is not till the capillaries become packed with corpuscles and the 

 circulation is confined to the arterial trunk that any rebound after 

 the ventricular contraction becomes apparent. This rebound is the 

 cause of throbbing in inflamed parts. 



The views here briefly given seem to me to form a consistent 

 theory, supported by experiment as far as the subject admits of ex- 

 periment, in accordance with the phenomena of inflammation as 

 observed in the warm-blooded animals. 



