498 Veterinary Medicine. 



Symptoms in the dog are those of hepatic congestion, or violent 

 gall stone colic, followed b}' severe rigor, great depression, or 

 irritabilit3^ and tenderness over the right hypochondrium. 

 Nansea and vomiting is a marked symptom though not a diag- 

 nostic one. 



Treatment. If the flaccid abdominal walls will allow of 'the 

 locating of the abscess it should be treated by aspiration and anti- 

 septic injections. It would even be admissible to perform lapar- 

 otom5^ stitch the wall of the -abscess to the external wound, and 

 empty it under due antiseptic precautions. 



INFECTKD HEPATITIS. NODUEAR NECROBIOSIS OF 



THE LIVER. 



In ox, sheep, pig, dog, horse. Necrotic areas projecting on surface of 

 liver. Causes : bacteria, toxins, from bowels, womb, navel. Lesions : In 

 cattle dirty gray nodules in brownish red liver, nodules firm, granular, ne- 

 crotic, elements do not stain, later leucocytes and fibro-plastic growth in 

 periphery. In lambs the nodules are white, common to the lungs and 

 pleura, pathogenic to rabbit. In pigs nutmeg liver, cells without nuclei, 

 fatty, granular, pathogenic to rabbits, guinea pigs, rats and 3'oung pigs. In 

 dog, nutmeg liver, with violet areas, and white spots, 1-2 lines, having 

 granular, fatty cells without nuclei. Symptoms : fever, constant lying, 

 tarry faeces, icterus, tender right hypochondrium, and those of the primary 

 disease. Treatment : antisepsis of primary seat, and bowels, elimination by 

 kidneys, general antisepsis, stimulants, etc. Case usually hopeless. Pre- 

 vention. 



This has been observed particularly in cattle, but also in sheep, 

 pig, dog and horse. It is characterized by the formation of cir- 

 cumscribed areas of gangrene, becoming hard, drj', yellowish 

 and usually slightly projecting beyond the adjacent surface. Its 

 infected character is .shown by the presence in the lesion and 

 adjacent parts of the hepatic tissue of an abundance of bacteria, 

 which, from the varied description, appear to differ in different 

 cases. The cause ma}' however be safely stated as one of the 

 bacteria of gangrene. It is alleged with some show of reason, 

 that the lesion mav be determined by the action of toxins and 



