Equine Influenza. Adynamic Catarrhal Fever of Solipeds. 127 



accompany those of pleurisy. The tumultuous heart-beats, often 

 associated with soft, weak or even rapid pulse, and later, a dead- 

 ening or muffling of heart sounds, as in hydropericardium are 

 characteristic when present. With endocarditis the early tumult- 

 uous heart-beats, with small weak pulse, irregular and sometimes 

 intermittent, become complicated by a blowing or hissing murmur 

 with the first heart-sound. In such cases clots of blood are liable 

 to form in connection with the valves, and may cause sudden and 

 early death. When the heart is involved the tendency to exten- 

 sive infiltration of limbs and lower aspect of the trunk is much 

 enhanced. (See diagnosis for table of phenomena in influenza, 

 fibrinous pneumonia and contagious pneumonia respectively). 



Symptoms of digestive disorder are usually in evidence. Even 

 in the thoracic forms the mouth is dry, hot, and has an offensive 

 odor ; the tongue coated above, has often red margins and tip ; 

 it may even be yellowish ; the gums may be swollen and dark 

 red or violet especially around the incisors ; mastication may be 

 slow and unwilling ; the pharynx maybe swollen, the pharyngeal 

 and submaxillary lymph glands may be tumid and tender, and 

 swallowing may be difficult. 



Congestions of the stomach and intestines are indicated by in- 

 appetance, sometimes flatulence, passage of flatus, constipation 

 with small, round, mucous-coated balls passed in small numbers, 

 and by slight transient colics, pawing, looking at the flanks, and 

 retraction of the abdomen. The retention of bile and destruction 

 of blood elements are indicated in a deeper yellow of the con- 

 junctiva and visible mucosae, and in a yellow, brown or red color 

 of the urine. There may be tenderness of the abdomen, but this, 

 like the colics, is moderate, the senses being blunted by the 

 attendant stupor which is usually even greater than in the 

 thoracic forms. Urination may become frequent with straining, 

 and the urine may become turbid, opaque, with flocculi of cystic 

 epithelium and mucus, and even albumen. In from three to five 

 days diarrhoea supervenes, the faeces becoming soft, pulpy, watery, 

 glairy or bloody, and escaping through a permanently dilated 

 sphincter. The diarrhoea may alternate with periods of torpor or 

 complete inactivity, otherwise tenesmus of the rectum is marked. 

 The exposed rectal mucosa is congested, of a deep red or 

 it may be of a dark violet hue. Eversion is not unknown. 



