Paludism in Dogs, etc. 577 



same principles that guide us in the ease of Texas fever. Search 

 should be made for an invertebrate host of the protozoon, by the 

 extinction of which infection may be stopped. The spring out- 

 break could be opposed by feeding hay on safe ground until the 

 higher pastures furnish sufficient vegetation. To counteract the 

 autumn attack, the sheep might be fenced out from the alkaline 

 bogs, and the forage supplied in the form of hay or soiling crops. 

 If it should appear that any wild animals harbor or transmit the 

 parasite, a campaign of extermination upon them would be in 

 order. If, as seems to have been the case in the early nineties, 

 the movement of sheep from the infected flocks and pastures 

 tends to cause the disease, this should be legally interdicted. 

 Finally, the complete extermination of the sheep on infected 

 areas could be practiced, and their place supplied by the immune 

 Angora goat. 



PALUDISM IN DOGS. MALIGNANT PROTOZOAN 

 JAUNDICE. 



Distribution : Senegal, Lyons, E. Africa, Paris, Pas de Calais, Cape 

 Colony. Microbiology : piroplasma : differentiation from that of Texas 

 fever : pathogenesis : tick-borne. Symptoms : incubation 3 to 5 days ; 

 dulness, prostration, apathy, drowsiness, anorexia, thirst, hyperthermia, 

 icterus, hemoglobinuria, offensive odor, emaciation, protozoon in globules, 

 loss of globules. Death in collapse. Lesions : body shrunken, emaciated, 

 fcetid ; dark tissues, mahogany yellow, petechise, enlarged congested liver 

 and spleen ; muco-enteritis ; bloody urine. Treatment unsatisfactory. 

 Prevention : keep from tick infested land ; clear and cultivate land ; smear 

 dog with insecticide ointment when hunting. 



In certain malarial districts dogs suffer severely and even 

 fatally from a febrile affection in which violent shivering is fol- 

 lowed by great hyperthermia and yellowish or brownish red dis- 

 coloration of the visible mucosae. It was frequently attributed 

 to malaria, and even sought to be identified with intermittent 

 fever in man. Marchoux in 1899 studied this disease on the 

 malarious seaboard of Senegal, and recognized the existence of 

 haemoglobinuria and the presence in the red globules of a heema- 

 tozoon. Leblanc, about the same date, found a protozoon in the 

 blood of dogs at Lyons, suffering from "red water," and Koch 

 37 



