THE PARASITE OF COAST FEVER 179 



forms escape into the blood-plasma, as a rule they 

 merely degenerate. 



While division by chromatin forking is character- 

 istic of the Babesia, it is not universal. Experience 

 has now shown that the organisms can divide into 

 two without the necessity for the somewhat com- 

 plicated chromatin forking. The nucleus simply 

 constricts into two, as in other Protozoa, the halves 

 separate, and the protoplasm follows the nuclear 

 division (Fig. 35, C-E). This has been seen in the 

 redwater parasite of Australia, in some English 

 cattle, and in the Babesia of the white rat. Briefly 

 stated, chromatin forking is common, but not 

 universal. 



The Protozoon responsible for Coast fever in cattle 

 presents considerable differences from that of red- 

 water. In the first place, not only does it infect the 

 red blood-corpuscles, but the leucocytes and the 

 glands may be infected. Gonder considers that 

 the multiplication is at its height in the glands, and 

 that it is uncommon in the peripheral blood. Hav- 

 ing had personal experience of the parasite (Theileria 

 parva), we can confirm the latter statement. 



The organism is very small and most difficult of 

 observation. It is also abnormally active, and can 

 change its place sometimes five or six times within 

 a minute, and executes a most irregular course 

 through the interior of the blood-corpuscle, a map 

 of its course somewhat resembling that produced by 

 superimposing several broken spider's webs. The 

 commonest forms seen in the blood are rodlike 

 (Fig. 37, D-F), club-shaped (Fig. 37, C), comma-like, 



