corn, canker, grease, laminitis, and punctured wounds of the foot. 

 Animals used for heavy draft, and those with flat feet and low heels, 

 are more liable to the disease than others, for the simple reason that 

 they are more exposed to injury. Rough roads also predispose to 

 the disease by increasing liability to injury. 



Symptoms. When the disease commences as a primary inflam- 

 mation of the cartilage, lameness develops with the formation of a 

 swelling on the side of the coronet over the quarter. The severity of 

 this lameness depends largely upon the part of the cartilage which is 

 diseased, for if the disease is situated in that part of the cartilage 

 nearest to the heel, where the surrounding tissues are soft and spongy, 

 the lameness may be very slight, especially if the patient is required 

 to go no faster than a walk ; but when the middle and anterior parts 

 of the cartilage are diseased, the pain and consequent lameness are 

 much greater, for the tissues are less elastic and the coffin joint is 

 more likely to become affected. 



- Except in the cases to be noted hereafter, one or more fistulous 

 openings finally appear in the tumor on the coronet. These openings 

 are surrounded by a small mass of granulations which are elevated 

 above the adjacent skin and bleed readily if handled. A probe shows 

 these fistulous tracts to be more or less sinuous, but always leading to 

 one point the gangrenous cartilage. When cartilaginous quittor 

 happens as a complication of suppurative corn, or from punctured 

 wounds of the foot, the fistulous tract may open alone at the point of 

 injury on the sole. 



The discharge in this form of quittor is generally thin, watery, 

 and contains enough pus to give it a pale yellow color ; it is offensive 

 to the sense of smell, due to the detachment of small flakes of car- 

 tilage which have become gangrenous and are seen in the discharge 

 as small greenish colored particles. In old cases it is not unusual to 

 find some of the fistulous openings heal at the surface; this is fol- 

 lowed by the gradual collection of pus in the deeper parts, forming 

 an abscess, which in a short time opens at a new point. The wall of 

 the hoof, over the affected quarter and heel, in very old cases becomes 

 rough and wrinkled like the horn of a ram, and generally it is thicker 

 than the corresponding quarter, owing to the stimulating effect 

 which the disease has upon the coronary band. Complications may 

 arise by an extension of the disease to the lateral ligament of the 

 coffin joint, to the joint itself, to the plantar cushion, and by caries 

 of the coffin 'bone. 



Treatment. Before recovery can take plare all of the dead car- 

 tilage must be removed. In rare instances this is effected by nature 

 without assistance. Usually, however, the disease does not tend to 

 recovery, and active curative measures must be adopted. The best 

 and simplest treatment, in a majority of cases, is the injection of 

 strong caustic solutions, which destroy the diseased cartilage and 

 cause its discharge, along with the other products of suppuration. 

 In favorable cases these injections will secure a healing of the wound 

 in from two to three weeks. While the saturated solution of sulphate 

 of copper, or a solution of 10 parts of bichloride of mercury to 100 



