Diagnosis. Treatment. Purulent Pneumonia. 145 



Diagnosis. The disease can be diag-nosticated witli cer- 

 tainty only after the microscopic detection of the moulds in 

 the secretions of the air passages, after the history and the 

 development of the disease have led to a suspicion of pneu- 

 monomycosis. Microscopic finding's are insufficient, because 

 similar moulds may be found in the air passages, particularly 

 of mammals, as harmless saprophytes. 



Treatment and Prophylaxis. To prevent the further multi- 

 plication of the moulds, inhalations with disinfecting solutions, 

 or intratracheal injections (see pages 42, 43) may be tried. 

 Their success appears very doubtful, because they do not reach 

 the moulds which are located in the alveoli. One might try the 

 methodical administration of iodide of potash. 



The most important measures are prophylactic. The animal 

 houses, especially those of fowls, must be kept clean and dry, 

 and the feeding of mouldy material must be avoided. To be 

 recommended are good ventilation, drying- out and cleaning 

 of the places, washing of the walls and wooden portions with 

 hot water or a disinfecting solution. 



Literature. Folger, Maanedsskr., 1906, XVIII, 311.— Johne, S. B., 1883, 52.— 

 Klee, Gefliigelkrkh., 1905, 49.— Liicet, De rAspergillus fumig., etc., 1897.— Lucet 

 & Constautin, Bull., 1906, 377.— Mace, Arch. <le Parasit., 1903, VII, 3 (with Lit.).— 

 Potain, Vet. Jhb., 1891, 190.— Eockl, D. Z. f. Tin., 1884, IX, 122.— Schiitz, Mitt, 

 d. G.-A., 1884, II, 208 (with Lit. ).—Thary & Lucet, Eec, 1895, 337. 



9. Purulent Pneumonia. Pneumonia suppurativa. 



{Abscess of the lung. Pneumonia emholica. Ahscessus pul- 



Etiology. A frequent cause of purulent pneumonia is the 

 lodgment of infected emboli, especially during the course of 

 septicopyemia of sucklings. Other sources of infected emboli 

 may be: purulent metritis after parturition, abscesses of 

 strangles, ulcerous endocarditis, purulent or gangrenous wounds 

 in various places of the body (pressure gangrene, gangrenous 

 inflammation of the feet), fistulas formed after venesection or 

 castration, etc. Foreign bodies, and with them pyogenic bac- 

 teria, more rarely get into the pulmonary tissue. Such foreign 

 bodies may enter the lungs from the air passages after injury 

 to the thoracic wall from the stomach. 



Suppuration of pulmonary tissue most rarely follows 

 croupous or catarrhal pneumonia (concerning catarrhal puru- 

 lent pneumonia caused by the bacillus pyogenes in cattle and 

 swine, see page 126). 



In some forms of catarrhal pneumonia or independently from it the bacillus 

 pseudotuberculosis of Preisz forms greenish yellow, soft caseous foci (see Vol. I), 

 and more rarely the bacillus necrophorus causes in catarrhal or croupous pneumonia 

 more dry and caseous foci. 



Anatomical Changes. In acute cases cavities are found 

 in the lungs filled with pus and tissue detritus; their internal 



