712 Compression of the Spinal Cord. 



Prog-nosis. This, as a general rule, is unfavorable, and 

 especially in large animals which are alreadj' unfit for work, 

 owing to somewhat severe motor disturbances, and in which 

 dangerous complications are likely to occur early. In the small 

 animals a complete recovery is not to be expected. Not rarely 

 the animals improve and the SJ^nptoms of paralysis disappear, 

 but the disappearance is only temporary, and, inasmuch as 

 the possibility of a regeneration is not to be expected, a re- 

 covery in the clinical sense cannot be hoped for. On the 

 grounds of the symptoms presented one cannot say whether 

 there is likely to be an improvement later or not, for this pur- 

 pose the animal must be under observation for a week or two, 

 during which time in favorable cases some improvement will 

 be noticeable. 



Treatment. No satisfactory treatment can be advised in 

 the case of the large animals. They should be slaughtered as 

 soon as possible. In the small animals the extirpation of neo- 

 plasms may be attempted, but in the majority of cases radical 

 treatment cannot be adopted owing to the impossibility of ex- 

 actly localizing the disease. In such cases there is not rarely 

 some improvement and even a complete disappearance of 

 symptoms if all movement be avoided and the animals kept in 

 a condition of complete rest. The animal's position should be 

 changed from time to time, and the urine and feces should be 

 removed. If the animals can move moderately well, slight move- 

 ments and massage of the muscles may be useful. Some au- 

 thors have observed improvement follow the injection of eserine 

 and pilocarpine, but such improvement might have happened 

 without any such treatment. 



Literature. Barrier, Bull., 1906, 283.— Dexler, (3. Z. f. \Ts., 1896, VII, 1; 

 Krgeb. (1. Path., 1896, III, 2, Abt., 516 (Lit.); 1900, VII, 479 (Lit.); Nerven- 

 krkh. d. Pferdes, 1899, 87 (Lit.).— Feuereissen, Z. f. Flhyg., 1905, XV, 86.— 

 Frohner, Monh., 1899, X, 123.— George & Johne, S. B., 1885, 40.— Goldmann, Z. f. 

 Flhyg., 1907, XVIII, 35.— Hamoir, Ann., 1904, 627; 1906, 332.— Haugmeier, Rep., 

 1853, 112.— Hink, D. t. W., 1899, 4.— Hinrichsen, A. f. Tk., 1882, XIV, 219.— 

 Jakob, M. t. W., 1910, 305.— Jakschatseh, B. t. W., 1899, 455.— Kammermann, 

 Schw., A. f. Tk., 1888, XXX, 205.— Nocard, Bull., 1885, 80.— Petit, Rec, 1906, 

 470.— Poes, Ann., 1902, 89.— Poulin, ibid., 1906, 687.— Rubay & Navez, ibid., 

 1902, 629.— Schmidt, A. f. Tk., 1889, XV, 295.— Stenstrom, Z. f. Tm., 1906, X, 

 113.— Tapken, D. t. W., 1905, 482.— Teetz, Z. f. Flhyg., 1905, XV, 60.— Wilson, 

 J. of comp. Path., 1904, 332. 



7. Syringomyelia. 



This name is applied in human medicine to a condition in which 

 there is an increase, and later a destruction of the neuroglia tissue with 

 the production of cavities, the Avails of which are formed of neuroglia 

 tissue. The cavities that are formed by dilatation of the lymph spaces, 

 or to softening of the tissues, observed in cases of myelitis, due to pres- 

 sure and in other forms of myelitis, are not included under the term. 



There are only two records of the occurrence of syringomyelia in 



