730 



A. S. WAKTHIN 



of this is slight. We know very little in fact of the pathologic processes 

 in this organ, even less than we do of its normal endocrin function. 



Fig. 11. Adenomatous new-formation of lobules of acinar tissue in chronic syphi- 

 litic pancreatitis with diabetes. Dilated acinar lumen. 



It is very probable that, until the physiologic problems regarding this 

 are solved, we cannot evaluate the pathologic changes occurring in the 

 organ or apply them to an explanation of the functional disturbance. It 



Fig. 12. High power view of new-formed acinar tissue seen in preceding figure. 

 Dilated lumen with vacuolated cells and hyperchromatic nuclei. 



must be constantly borne in mind that the islet changes seen at autopsy in 

 the diabetic are an end-result. The hyaline islet tells us no more about the 

 essential disturbance of the islet cells than do the hyaline glomeruli of the 



