850 



TABLE VI 

 TOXEMIA, NEPHRITIC TYPE. (No CONVULSIONS.) (Caldwell and Lyle) 



(NOTE. Figures in italics represent the control test 4 days or more post partum. ) 



is typical of chronic nephritis but the figures of Case 7 show less reten- 

 tion than is usually found in uremia and are similar to those of convulsive 

 eclampsia. 



This table offers direct evidence of the value of blood determina- 

 tions for diagnosis and also for prognosis. The trend of the figures is in 

 relation to the severity of the clinical symptoms. 



In 12 typical eclamptic cases, it was found that they all had albumin 

 and casts in the urine and a high systolic blood pressure. Eight of the 

 cases recovered and therefore the observer was able to procure more than 

 one blood specimen in most instances. The first three cases show very 

 slight changes in the partitions. Case No. 1, Table VII is quite normal 

 except for the rise in the uric acid. The next two cases have, in addition 

 to the increase in the creatinin and uric acid, a slight rise in the urea. All 

 the others have an increase in the figures, but not as marked as is 

 usual in chronic nephritis, except Case No. 9 which has a retention of the 

 urea. The urea figures in Caldwell and Lyle's cases are somewhat higher 

 than those presented by Siemens and by Losee and Van Slyke and are 

 more in accord with those of Fair and Williams. As many cases on recov- 

 ery show higher figures than others do at the time of their convulsions; 

 and as many chronic nephritic cases show higher figures without any 

 increase in their clinical symptoms, it becomes evident that these changes 

 are the result rather than the cause of the toxemia leading to eclampsia. 

 Of seven of the eclamptic cases that recovered a follow-up examination 

 after two years shows that Cases 3 and 6 are normal from a clinical stand- 



