TxENEKAL SYMPTOMS. 65 



be good, the fever is characterised by much heat, and a strong, 

 full pulse; on the contrary, if the animal's constitution be 

 impaired, or if it be out of condition, the fever is of a low 

 type, called typhoid, asthenic, or adynamic, and is charac- 

 terised by prostration, sinking of the pulse, great impairment 

 of the heart's action, with a tendency to collapse ; and the skin 

 is covered by cold, clammy sweats. Sometimes these adynamic 

 characters pass into that typhoid state in which nervous symp- 

 toms, such as delirium, coma, and tremors, prevail. These 

 characters are known as nervous or ataxic. There is uncon- 

 sciousness, grinding of the teeth, the mouth becomes foetid, the 

 tongue dry, and tremors affect the voluntary muscles. This 

 form sets in as a result of some unhealthy tendency of the 

 inflammatory process, or when mortification of the inflamed 

 part occurs ; but any cause by which the system becomes 

 vitiated will bring about this kind of fever without death 

 of the inflamed part, such as the poison of rinderpest, acute 

 glanders, and other allied diseases. Putrescence of the pro- 

 ducts of inflammation, degenerating and decomposing, poison 

 the fluids circulating amongst them, and when absorbed induce 

 the typhoid state. This form of fever tends to death by com- 

 plete sinking of the circulation, with diminution and loss of 

 animal heat ; or deepening stupor, with oppressed respiration, 

 supervenes ; or the animal dies by combination of both asthenia 

 and coma. 



Be it understood, however, that sympathetic inflammatory 

 fever is especially marked by the absence of certain symptoms 

 which distinguish the idiopathic fever, more especially of 

 petechia, and of any special eruption on the sldn. 



INFLAMMATORY APPEAEANCES OF THE BLOOD. 



The chief appreciable change in the constitution of the blood 

 is a great increase in the quantity of its fibrine, which, accord- 

 ing to Andral and others, may range from 2^ to 10 per 1000 

 parts. It is, however, a very difficult matter to ascertain with 

 any certainty the exact increase, for it is impossible, by any 

 process yet discovered, to separate the fibrine from the white 

 corpuscles of the blood. 



Some pathologists state that there is an increase of the 

 P 



