INFECTIOUS TRACHITIS 29, 



li-.wered and t'-.e beak allowed to re-t on the hreaj-t for a few moment,-. 

 Tlie eomi) and wattles were cyanotic in appearance. Tiie Iiead find 

 legs felt cold to tlie touch. Since the projrress was unfavorable 

 the bird was killed. The general condition of the cockerel was 

 emaciated and anemic. Necropsy revealed patches of yellowish-white 

 pseudomembrane in the pharynx, while the larynx was nearly occluded 

 witii a cheesy white mass. The trachea showed des(]uaniative epithelial 

 inflammation and hemorrha.ffes of the mucosa and submucosa. In addi- 

 tion, fatty de,£i:eneration of the liver and hemorrhagic inflammation of 

 the tubular tissues of the kidneys were present. Histopathological sec- 

 tions of tissues from the trachea showed desquamation of the epithelium, 

 hcmorrhaaes and coagidation necrosis of the mucosa and submucosa. 



Cockerel ^'P SSI did not show any symptoms of disease for nine days. 

 On November 29. it was not as active as usual, the feathers appeared 

 ruffled, saliva flowed from the beak, and all desire for feed had vanished. 

 The bird was weak, scarcely able to stand, and dyspnea uas apparent. 

 Without showina- any sii>:ns of iiuprovement the cockerel died two days 

 later. The carcass was emaciated. The comb and wattles were cyanotic. 

 Postmorteiu examination revealed the presence of con.trestion and pus 

 in the larynx and posterior nares. The trachea was filled with a purulent 

 muco-fibrinous mass, and the mucosa was inflamed and necrotic in places. 

 The lungs were congested and showed consolidated areas near the periphery. 

 Histopathological slides ]irepared from diseased tissues showed hyperemia 

 and fibrino-purulent inflammation of the tracliea and larynx, congestion 

 of the lungs with a large number of small mononucle;ir leucocytes in the 

 alveolar spaces, cloudy swelling and fatty degeneration of the liver, and 

 cloudy swelling and hemorrhagic inflammation of the tubular areas of the 

 kidneys. 



Cockerel ^'P S82 showed symptoms after six days. On November 20 

 the bird iiad no desire for feed, appeared to be weak, the comb and wat- 

 tles were cyanotic, and there was a j^ronounced inspiratory dysi>nea. The 

 cockerel died the following day. The general condition of the carcass 

 was fair. Necrojjsy revealed destjuamative inflammation of the trachea. 

 The exposed surface of the mucosa appeared dry and of a grayish-yellow 

 color. The syrinx was occluded with dried exudate containing desquamat- 

 ed ciliated epithelium, apparently from the trachea. The lungs, kidneys, 

 liver, and spleen were hemorrhagic in appearance. Histological sections 

 of tissues from the diseased areas, stained by Unna's alkaline methylene 

 blue and eosin, confirmed all of the gross findings. 



In addition, the trachea showed hyperemia and fibrinous inflammation 

 of the mucosa and submucosa; the lungs, interstitial inflammation with a 

 tendency toward consolidation at the peri])hery; the kidneys, cloudy swell- 

 ing; the liver, perivascular foci of hyperplasia of histiocytes and hydropic 

 degeneration; the spleen, congestion and large number of polymorphonu- 

 clear and small mononuclear leucocytes in the arteries, capillaries, and in 

 some places in the parenchyma. 



Cockerel VP 884 showed symptoms of disease eight days after inocu- 

 lation. On November 28 this cockerel did not eat, the feathers appeared 

 ruffled, and the face was pale. The next day dyspnea and emesis were 

 present. The vomiting was apparently caused by fermented feed in the 

 crop. An examination by transmitted light disclosed pseudomembrane 



