82 



SWINE PLAGUE 



cytes, and hence the gradual cutting off of the blood supply. 

 One is a rapid death due directly to highly virulent bacteria, 

 the other a slow death, or a kind of dry suppuration in the 

 later stages of the pneumonia, characteristic of the pig, and 



due indirectly to the 

 irritation of perhaps 

 more attenuated races 

 of bacteria. In some 

 cases there are exten- 

 sive hemorrhages in 

 the interlobular con- 

 nective tissue. 



The infllamraation 

 of the pleura fre- 

 quently extends to the 

 pericardium. This 

 membrane is opaque, 

 thickened and its ves- 

 sels distended. It 

 may be glued to the 

 contiguous lobes of 

 the lungs and covered 



by a false membrane, smooth or roughened, which extends 

 upon the large vessels emerging at its base. 



Disease of the digestive tract in a considerable proportion 

 of animals inoculated with swine-plague cultures consisted 

 n a severe catarrhal inflammation of the lining membrane 

 of the stomach. The hyperemia was very intense, bordering 

 on hemorrhage. Occasionally the extension of the peritonitis, 

 produced by intra-abdominal inoculation along the mesentery, 

 causes a severe inflammation, with exudation on the mucosa 

 of the small intestine. A case is reported where all the Peyer's 

 patches of the small intestine were in a hyperemia and partly 

 hemorrhagic condition. 



In the naturally contracted disease extensive hyperemia 

 of the mucosa of the large intestine, bordering on a hemor- 

 rhagic condition, has been observed. In other cases a peculiar 



Fig. 12. Hemorrhage in the interlobular 

 tissue of a sivine-plague lung: (a) hemor- 

 rhage, {b) hepatized lobules. 



