HEREDITY. 





Table 11a.— Distribution of prom ny of the different maHngt according t>> index of build; i 



andfemalee tabulated separately— Continm d 



Avg. 9 . 34.54 ±0.17 S. D. 9 , 5.133 ± 0.089 



The one clear conclusion from this study is ilia! no MH2.lt* diai 

 and no special single collection of disease- i- exclusively responsible 

 for exceptionally slender or except idiiallv fleshy build. The variation! 

 in build are due primarily rather to various idiosyncrasies of develop- 

 ment and metabolism which have largely an hereditary basis, upon 

 which may be superimposed modifications by various types of dig) :i-» 



