326 HISTOLOGY OF THE MORBID PROCESSES. 



From the examples that have been given it will be noticed that, 

 amid all its protean manifestations, the inflammatory process is fun- 



FIG. 288. . 

 a b' b 

 | I I 



L^ x ;i*Sl*tar4l 

 I* 



si 









Chronic interstitial myocarditis, late stage : a, dense fibrous tissue, the final result of the 

 interstitial inflammation ; 6, b', b", atrophied cardiac muscle-cells ; b', vacuolation of a 

 less atrophic coll ; b", section showing anastomotic branch joining two cells ; c, partially- 

 obliterated bloodvessel. 



damentally the same, but susceptible of many variations ; and when 

 the conditions are not too adverse it leads to a removal of the cause 

 of an injury and to a more or less complete repair or patching of 

 the tissues that have been damaged. 



III. INCIDENTAL CONSEQUENCES OF DAMAGE AND 

 INFLAMMATION. 



The damage and ensuing inflammation affecting a part of the 

 body not only occasion changes in the structure of that part, but 

 also, through those changes, very frequently cause morbid conditions 

 in remote parts. It will be impossible to enumerate all the possi- 

 bilities in this connection, but a few examples will suffice to show 

 their importance. It is obvious that chronic interstitial hepatitis 

 (Fig. 286) must affect the circulation in the portal system of vessels. 

 The inflammatory fibrous tissue formed between the lobules of the 

 liver, and, therefore, around the portal vessels within that organ, pos- 

 sesses the same tendency to contract after its formation that is mani- 

 fested by cicatricial tissue of more acute inflammations, though perhaps 



