Drupaceous Fruits 75 



CHERRY 



Leaf-spot ^' ^^~^^ {Coccomyces sps., Cylindrosporium) . — 

 The disease, first reported in Europe in 1884, is widespread 

 wherever cherries are cultivated and is usually very destruc- 

 tive. Sweet cherries are most susceptible. In Ohio the loss 

 in one year was estimated at $25,000. This disease, on fruit, 

 pedicels, and leaves is nearly identical in symptoms with 

 that of leaf -spot of the plum. For description see p. 98. 

 Diseased refuse, leaves, etc., should be plowed under or 

 destroyed. Sour cherry trees should be sprayed with lime- 

 sulfur solution diluted at the rate of 13^ gallons to 50 gallons 

 of water, or with 3-4-50 Bordeaux mixture: (1) as soon as the 

 petals have fallen, (2) about three weeks later, (3) directly 

 after the fruit is picked. Arsenate of lead, 1 pound of powder 

 to 50 gallons of spray, may be added for the control of insect 

 pests. 



Sweet cherries should receive the same treatment as the 

 sour except that lime-sulfur solution diluted at the rate of 

 1 gallon to 50 gallons of water should be the fungicide used. 

 Bordeaux mixture should never be used on sweet cherry 

 trees, because of the risk of injury. Dusting has proved 

 satisfactory in some localities. The results of spraying by 

 Scott are shown in the accompanying figures. These trees, 

 located in Illinois, were sprayed three times. 

 -Black-knot (Dihotryon morhosum). — ^As upon the plum, 

 this knot causes serious injury to the cherry. In some 

 sections it is so prevalent on the wild cherry and plum 

 trees as to render control practically impossible, and in 

 certain regions has caused the abandonment of the cherry- 

 growing industry. Taken in time it is easy to control. For 

 treatment see plum. 



Brown-rot, mold {Sderotinia cinerea, Monilia). — -The 

 same fungus which produces disastrous results upon the 

 peach also causes the most serious disease of the cherry, being 

 especially destructive on sweet cherries. Several stages of 

 its development are illustrated in Fig. 38. The disease is 



