UDDER-ACTINOMYCOSIS 113 



lymphogenous metastasis and by infection through the teat 

 canal. In one case of distinctly primary udder-actinomy- 

 cosis induced by teat-infection all four quarters became 

 affected. 



Primary udder-actinomycosis following wound infection 

 is a condition I have not seen, but if it occurs it certainly 

 has a particular anatomical character which closely simu- 

 lates the corresponding type of udder tuberculosis. 



Secondary or embolic udder-actinomycosis is a condition 

 I have not observed in any of the udders that I have ex- 

 amined, but that it can occur appears to be natural. 



The possible secondary infections might be strepto- 

 and staphylomycosis, colibacillosis and perhaps pyo- 

 bacillosis. 



Secondary infection through wounds always occurs 

 through an actinomycotic wound, and here, as with all 

 chronic wounds, secondary infection is the rule. Secondary 

 infections which one may expect in this case are the same 

 as those previously described. 



Saprophytic invasion can induce milk- and wound- 

 gangrene. 



Milk-gangrene [gangrenous mastitis] does not appear 

 to occur in primary udder-actinomycosis. It is possible in 

 secondary infections. 



Wound-gangrene, when it occurs, always develops from 

 an actinomycotic wound, and it may easily develop in this 

 condition. As previously stated, a wound of this kind is 

 very infrequent. 



General Conditions. — Infection of the blood with 

 metastasis to the lungs is an uncommon complication. 



Intoxication. — Udder-actinomycosis usually presents 

 no distinct general symptoms. If the condition is far ad- 

 vanced, however, or metastatic processes have developed the 

 animal may emaciate nevertheless, and finally present 

 symptoms of anemia and debility. 



