Glanders. 



317 



the cat family the hing's arc involved just as in croupous pneumonia 

 and show stages of red and gray hepatization. In the horse the 

 lungs are apt to show a confused mixture of inflammatory changes ; 

 groups of lobules are found all through the lungs forming wedge- 

 shaped, swollen areas of a densely elastic consistence cutting like 

 bacon, mottled reddish-yellow or grayish-yellow and leaving on the 

 knife blade a turbid muco-purulcnt juice. In tlie chronic type the 

 pulmonary tissue is changed into an extremely tough, light grayish- 

 yellow mass of connective tissue riddled with confluent cavities about 

 as large as a thumb's breadth, filled with a slimy, oily pus and having 

 a deeply reddened inner surface. In this type of change the lymph 



%;\>^" 



Fig 79. 

 Large glanders ulcer of nasal septum of horse. 



glands become very large and swollen, soft like marrow ; afterwards 

 densely indurated and changed into a tough white connective tissue, 

 forming nodular bunches as large as a goose tgg which become ad- 

 herent to the surrounding structures by similar fibrous bands. In 

 the interior of these hyperplastic and inflamed lymph glands is 

 encountered either a purulent softening in isolated spots or exten- 

 sive purulent liquefaction with cavity formation, or an indurated, 

 drv cheesy necrotic focus. The multiplication of the glanders 

 bacilli in the lymph paths, besides causing them to swell up like 

 cords, also gives rise to a diiTuse gelatinous infiltration (especially 

 because of the lymph stagnation), and, if the inflammation become 

 chronic, to broad connective tissue thickenings ; so that, for example, 

 the loose cellular tissue of the skin becomes the seat of gelatinous 

 thickening with extensive indurations all through it (pachydermia, 

 elephantiasis) with foci of suppurative necrotic destruction running 

 all through the skin and subcutaneous tissue. 



