and ZnMet had higher liver Zn than controls. Muscle Cu was 

 highest for controls. For EXP 2, organic Zn sources were 

 equally or more BAV than the best inorganic source. In EXP 3, 

 four sheep were administered 250 mg of SUP Cu from either 

 CuLys or CuS0 4 . One of the CuS0 4 treated sheep was very 

 sensitive to Cu and the other was not affected by Cu excess. 

 Myodegradation was present in one animal from each T prior to 

 death. For EXP 3, the development of toxicity was not 

 affected by source. In EXP 4, 63 rats were given ZnMet, 

 ZnLys, or ZnS0 4 (30 mg Zn/kg) and CuLys, CuS0 4 , or CuO (6 mg 

 Cu/kg) in a 3 x 3 factorial EXP. After 4 wk, four rats from 

 each T were sacrificed and the remaining rats fed a non- 

 supplemented diet for 1 wk . Plasma Cu was lower for animals 

 supplemented with CuO than CuS0 4 and CuLys. Bone Zn was 

 higher for CuLys than CuO. The CuO T was lowest in BAV and 

 kidney. Rats supplemented with CuS0 4 had higher muscle Cu 

 than with CuLys. After depletion, plasma Cu was lower for CuO 

 than CuLys, kidney Zn lower for CuS0 4 than for CuO which in 

 turn had the lowest liver Cu . Amino acid complexed minerals 

 were highly available. They were equal to and in some cases 

 higher in BAV than the sulfate form and considerably more 

 available in most cases than the oxide forms. 



XI 1 



