SECRETION PHYSIOLOGY. 317 



Experiment Va (Continued; see p. 305). 



Time. Nerve. Secretion in mm. 



h. m. s. 



3 25 Artery clamped close by the hilus. 



3 30 ..... , Chorda o 



3 35 S}Tiipathetic 23 



3 37 Sympathetic o 



3 40 Sympathetic o 



0.2 cc, .5 % NaCl solution injected into duct. 

 3 41 Sympathetic • . 17 



3 42 Artery undamped 



4 07 30 Artery clamped 



4 12 Chorda o 



4 13 Sympathetic 25 



4 15-4 17 ... . Chorda o 



4 17 30-4 18 15 . Sympathetic 14 



4 20 Sympathetic o 



4 23 . . . . . . . .3 cc, .5 % NaCl injected into duct 



4 24 Sympathetic .... .... 13 



4 25 Sympathetic o 



4 26-4 27 .... Sympathetic o 



4 28 2 cc, .5 % NaCl injected 



4 29 Sympathetic 8 



In this experiment the sympathetic appeared paralyzed at 

 3:40, 4:20 and 4:26, but the injection of normal salt solution 

 into the duct was followed by a secretion little less than normal, 

 on the next stimulation. In one case twenty minutes after the 

 artery had been clamped, the sympathetic was thus shown still 

 to be active. Heidenhain attributes the loss of the chorda's 

 power to the suffocation and consequent paralysis of the gland 

 cell. (See footnote, p. 308.) As already pointed out (p. 3 16) this 

 would, if true, show that the sympathetic produces its secretion 

 in some other way than by action on the cell. The fact that 

 the nerve's power may be restored by the injection of innocuous 

 fluid into the ducts is readily explicable on the muscular theory 

 of secretion, but, with difficulty, on the cellular theory. 



I found that a similar phenomenon may, at times, be seen in 

 the cat's submaxillary, which has been paralyzed by just suffi- 

 cient atropin to prevent chorda secretion. As was first pointed 

 out by Langley, atropin paralyzes the sympathetic in the cat, 

 but more atropin is required than to paralyze the chorda. The 



