3388 The Philippine Journal of Science 1915 
TABLE J.—Record of experiments with fowls. 








{ Pe ea| ‘| 
No.of | Gondition. hexper Ween | Day ctdcan | Sain aa 
| i | = 
| Days. | Per cent. | | 
1 | White rice control___-|__________ Ah) Ab anaes tr | Typical neuritis. 
iene (0 Ko peers eevee yo lee ee | —29.9 | Twenty-ninth _______- Do. 
G} es Coe s-Ses ead. Bee See ee —21.7 | Twenty-second _______ Do. 
Aue (o Reena ee ae Bh o | es ana —40.8 | Twenty-eighth_______ Do. 
6 || Palay control__-_-_--~ | GORA ect AO) es Je Ae oe eee eee Alive and well at 
| | end of 60 days. 
Gilead Oe aes sane res 60 | Lee e tse oe somes. s eves os! Do. 
(vier dortess: eae 60 a ek eee eae ee See OE oS Do. 
yea do MARRS gt | G0 HOLE OU ELNAL AN LLene eee Do. 
9 | White rice and 40 |-------___ et 2120)|(Seventhi=s= e222 es Apparent exhaus- 
minutes exercise. | | | tion after exer- 
1 cise. 
No neuritis. 
I) Wasa Mo: -S222si Gites tly oeree cies ——14-on et Ourth =. oases eee | Do. 
dt SWihitemericessand sz) eee Sabyas || Uberti Se | Typical neuritis. 
minutes exercise. | 
1) lho a ig 2 SERS Tet SES —20.2 | Twenty-fourth _______ Do. 
13 | Palay and 40 minutes 60 == 16!,0" | es Se ER oe wah | Alive and well at 
exercise. end of experi- 
| | ment. 
Any ene Oe Sie eT Ae GO|) = GEG cocceececneesessaseae ss) Do. 
15 | Palay and 20 minutes 60 = D186 )|/se 5 Se. eae se 2 Do. 
exercise. { 
Go| eee (s (oye en hs oe 60 == 10 Ol | Semen en a eee Do. 
Ia eWihitemricelatiar cee. sae See || aaa ee Prostration with- 
out neuritis. 
18) [ eee 1 eae ene Sees, ee Bee SE Sy qraai kd) oo Typical neuritis. 
(70) 19 |____- (0 (oye meres SOE S| a ei 64.2 | Thirty-third—_______-- Do. 
C73) e201 | eaeee CORSE See 8 ee ee —— 3410) een une ee se wae ee ee ee | Prostration and 
| | slight neuritis. 
(Al) PAN |] TAAENY ans BIO 2 -- = 60 =| eee ee Alive and well at 
| end of 60 days. 
(92) 422) |2e2e2 Goeeeaaee ea eeee 60 ==. 838 || 22Et sce. cee taeee = Sa | Do. 
23) Wihiteinice atl5o (222) -2ee ee SSRIS) AN ana eee Typical neuritis. 
Bo | cece KY gu ae eee a aes —21.5 | Forty-fourth --.-.---- Do. 
| 




That beriberi is due to an intoxication of some sort has been 
the basis of several theories of the etiology of the disease. Such 
a hypothesis has much support in the symptom complex, espe- 
cially of acute beriberi. Very similar forms of neuritis are 
produced by alcohol, arsenic, and some other poisons and a few 
toxins. Since the discovery of the vitamines there has been a 
general tendency to ignore this evidence and accept the idea that 
beriberi is due to a deficiency of vitamines per se. A theory 
that regards the vitamines simply as constituents necessary for 
tissue building fails to account rationally for a number of re- 
corded facts regarding beriberi in men and animals. 
